Back to search

Article

Modulation of Host Cell Death Signaling Platforms by <em>Plasmodium falciparum</em>: Implications for Eryptosis Regulation and Parasite Survival

2026-08-17

Abstract excerpt

Programmed cell death pathways in Plasmodium falciparum remain conceptually fragmented. Over decades, researchers have applied metazoan apoptotic, autophagic, and necrotic markers to this deep-branching protozoan, frequently clashing with the reality that the parasite lacks the canonical genetic machinery (such as true caspases or death receptors) found in multicellular eukaryotes. In this work, we shift the focus...

Topics

Open a Topic to create a Post that cites this publication.

Identifiers and source

Literature Corpus work
1f9766c0-1995-53e4-bde0-410e6398f3ca
DOI
10.20944/preprints202608.1100.v1
Open publication

Related research

Semantic proximity does not establish scientific evidence.

Click a neighbor to travelStep 1 · 12 closest
Interactive article relationship graphSelect a related publication card to move it into the centre and load its closest explainable connections. Solid lines are source-backed structured connections. Dashed lines are semantic discovery signals and are not scientific evidence.
Modulation of Host Cell Death Signaling Platforms by <em>Plasmodium falciparum</em>: Implications for Eryptosis Regulation and Parasite SurvivalDOI 10.20944/preprints202608.1100.v1
Manipulating Eryptosis of Human Red Blood Cells: A Novel Antimalarial Strategy?Semantic proximity
Proteomic and genetic analyses demonstrate that Plasmodium berghei blood stages export a large and diverse repertoire of proteins.Semantic proximity
Moving on: How malaria parasites exit the liverSemantic proximity
Management of cell death in parasitic infectionsSemantic proximity
Erythrocyte remodeling by malaria parasitesSemantic proximity
Maurer's clefts, the enigma of <i>Plasmodium falciparum</i>Semantic proximity
Chloroquine induces eryptosis in <i>P. falciparum-infected</i> red blood cells and the release of extracellular vesicles with a unique protein profileSemantic proximity
Plasmodium berghei EXP-1 interacts with host Apolipoprotein H during Plasmodium liver-stage developmentSemantic proximity
Characterisation of Two <i>Plasmodium</i> Virulence Factors Important for Lipid Metabolism and Disease Progression <i>In Vivo</i>Semantic proximity
<i>Plasmodium falciparum</i> diacylglycerol acyltransferase maintains phospholipid homeostasis to regulate sexual differentiation, ER stress, and cytoadhesionSemantic proximity
MMV687794 blocks <i>Plasmodium falciparum</i> invasion of red blood cells by targeting a Surface-associated Lipid-Interacting Rhoptry Protein, <i>Pf</i> SLIRPSemantic proximity
The long and winding road: Protein trafficking mechanisms in the Plasmodium falciparum infected erythrocyteSemantic proximity
Select a neighboring publication to make it the new centre.