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Article

Structural Organization of the Human Flavin-Containing Monooxygenase 3 Gene (FMO3), the Favored Candidate for Fish-Odor Syndrome, Determined Directly from Genomic DNA

1 Dec 1997

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 9417913
DOI: 10.1006/geno.1997.5031

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Structural Organization of the Human Flavin-Containing Monooxygenase 3 Gene (FMO3), the Favored Candidate for Fish-Odor Syndrome, Determined Directly from Genomic DNAPMID 9417913Read ↗

Mutation, polymorphism and perspectives for the future of human flavin-containing monooxygenase 3.Semantic proximity Read ↗

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Identification and functional analysis of common human flavin-containing monooxygenase 3 genetic variants.Semantic proximity Read ↗

Missense mutation in flavin-containing mono-oxygenase 3 gene, FMO3, underlies fish-odour syndrome.Semantic proximity Read ↗

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Simple confirmation methods for rare but impaired variants of human flavin-containing monooxygenase 3 (FMO3) found in an updated genome resource databank.Semantic proximity Read ↗

Molecular cloning, sequence characterization, SNP detection, and tissue expression analysis of duck FMO3 gene.Semantic proximity Read ↗

A novel mutation in the flavin-containing monooxygenase 3 gene (FMO3) of a Norwegian family causes trimethylaminuria.Semantic proximity Read ↗

Targeted sequencing identifies genetic polymorphisms of flavin-containing monooxygenase genes contributing to susceptibility of nicotine dependence in European American and African American.Semantic proximity Read ↗

Discovery of novel flavin-containing monooxygenase 3 (FMO3) single nucleotide polymorphisms and functional analysis of upstream haplotype variants.Semantic proximity Read ↗

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