Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Examination of two genetic polymorphisms within the renin-angiotensin system: no evidence for an association with nephropathy in IDDM.

Diabetologia - 1 Sept 1996

Chowdhury T A, Dronsfield M J, Kumar S, Gough S L, Gibson S P, Khatoon A, MacDonald F, Rowe B R, Dunger D B, Dean J D, Davies S J, Webber J, Smith P R, Mackin P, Marshall S M, Adu D, Morris P J, Todd J A, Barnett A H, Boulton A J, Bain S C

Abstract excerpt

Premature cardiovascular disease is common in insulin-dependent diabetic (IDDM) patients who develop diabetic nephropathy. Genetic polymorphism within the renin-angiotensin system has been implicated in the aetiology of a number of cardiovascular disorders; these loci are therefore candidate gene...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Age of Onset
Share to Alleles

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Cohort Studies

Share to Diabetes Mellitus, Type 1

Share to Gene Frequency

Share to Genotype

Share to Polymorphism, Genetic

Share to Probability

Source

Identifiers

PMID: 8877296
DOI: 10.1007/BF00400661

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026. Prepared record publication-8877296-2026-09-01.1. Manifest publication-graph-cohort-2026-09-01.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 1 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Examination of two genetic polymorphisms within the renin-angiotensin system: no evidence for an association with nephropathy in IDDM.PMID 8877296Read ↗

Evaluation of risk factors for the development of nephropathy in patients with IDDM: insertion/deletion angiotensin converting enzyme gene polymorphism, hypertension and metabolic control.Semantic proximity Read ↗

Lack of association of angiotensin II type 1 receptor gene polymorphism with diabetic nephropathy in insulin-dependent diabetes mellitus.Semantic proximity Read ↗

Association of ACE gene polymorphism and diabetic nephropathy? The Diabetic Nephropathy Study Group.Semantic proximity Read ↗

Genetic variants of the renin-angiotensin system, diabetic nephropathy and hypertension.Semantic proximity Read ↗

Angiotensinogen gene polymorphisms in IDDM patients with diabetic nephropathy.Semantic proximity Read ↗

Angiotensin converting enzyme (ACE) insertion/deletion (I/D) polymorphism, and diabetic retinopathy in subjects with IDDM and NIDDM.Semantic proximity Read ↗

Genetic predisposition to nephropathy and associated cardiovascular disease in people with type 1 diabetes: role of the angiotensinI-converting enzyme (ACE), and beyond; a narrative review.Semantic proximity Read ↗

Angiotensin I converting enzyme gene polymorphism and diabetic nephropathy in type II diabetes.Semantic proximity Read ↗

Lack of relationship between an insertion/deletion polymorphism in the angiotensin I-converting enzyme gene and diabetic nephropathy and proliferative retinopathy in IDDM patients.Semantic proximity Read ↗

Angiotensin I-converting enzyme and angiotensinogen gene polymorphisms in non-insulin-dependent diabetes mellitus. Lack of relationship with diabetic nephropathy and retinopathy in a Caucasian Mediterranean population.Semantic proximity Read ↗

Investigation of ACE, ACE2 and AGTR1 genes for association with nephropathy in Type 1 diabetes mellitus.Semantic proximity Read ↗

Lack of association between the insertion/deletion polymorphism of the angiotensin-converting-enzyme gene and diabetic nephropathy in IDDM patients.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.