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Article

Testicular and Ovarian Resistance to Luteinizing Hormone Caused by Inactivating Mutations of the Luteinizing Hormone–Receptor Gene

22 Feb 1996

Abstract excerpt

In normal males, luteinizing hormone (LH) regulates the function of Leydig cells and, hence, male sexual differentiation, pubertal androgenization, male sexual function, and fertility. Abnormalities in the function of Leydig cells result in primary hypogonadism and varying degrees of male pseudohermaphroditism.1-5 In these patients, Leydig cells are absent, hypoplastic, or unresponsive to stimulation with human...

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Identifiers

PMID: 8559204
DOI: 10.1056/nejm199602223340805

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Related research

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Testicular and Ovarian Resistance to Luteinizing Hormone Caused by Inactivating Mutations of the Luteinizing Hormone–Receptor GenePMID 8559204Read ↗

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Menstrual disorders and infertility caused by inactivating mutations of the luteinizing hormone receptor gene.Semantic proximity Read ↗

Luteinizing hormone resistance syndromes.Semantic proximity Read ↗

[Inactivating and activating mutations of the human LH/hCG receptor leading to male pseudohermaphroditism and familial male-limited precocious puberty].Semantic proximity Read ↗

Activating and inactivating mutations in LH receptors.Semantic proximity Read ↗

Inactivating mutations of luteinizing hormone beta-subunit or luteinizing hormone receptor cause oligo-amenorrhea and infertility in women.Semantic proximity Read ↗

Mutations along the hypothalamic-pituitary-gonadal axis affecting male reproduction.Semantic proximity Read ↗

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