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Article

A multitude of suppressors of group II intron-splicing defects in yeast.

Current genetics - 1 Oct 1993

Waldherr M, Ragnini A, Jank B, Teply R, Wiesenberger G, Schweyen R J

Abstract excerpt

Disruption of the nuclear MRS2 gene (mrs2-1 mutation) causes a strong pet- phenotype in strains with mitochondrial group II introns, and a leaky pet- phenotype in strains without group II introns. MRS3 and MRS4, the genes for two mitochondrial-solute carrier proteins, can suppress both phenotypes when present in high-copy-number plasmids. In order to search for further multicopy suppressors of the mrs2-1 mutant...

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Source

Identifiers

PMID: 8252639
DOI: 10.1007/BF00336780

Genes

MRS2

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

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A multitude of suppressors of group II intron-splicing defects in yeast.PMID 8252639Read ↗

The nuclear gene MRS2 is essential for the excision of group II introns from yeast mitochondrial transcripts in vivo.Semantic proximity Read ↗

Mutant alleles of the MRS2 gene of yeast nuclear DNA suppress mutations in the catalytic core of a mitochondrial group II intron.Semantic proximity Read ↗

MRS3 and MRS4, two suppressors of mtRNA splicing defects in yeast, are new members of the mitochondrial carrier family.Semantic proximity Read ↗

Mitochondrial Mg(2+) homeostasis is critical for group II intron splicing in vivo.Semantic proximity Read ↗

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A member of a novel Arabidopsis thaliana gene family of candidate Mg2+ ion transporters complements a yeast mitochondrial group II intron-splicing mutant.Semantic proximity Read ↗

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