Deletions of Tn916‐like transposons are implicated in <i>tetM</i>‐mediated resistance in pathogenic <i>Neisseria</i>

Abstract excerpt

Using the tetM-containing conjugative transposon Tn916 as a mutagenesis tool, we identified two distinct classes of transposon insertions in the meningococcal chromosome. Class I insertions have an intact copy of Tn916 that appears to have transposed by a novel recombinational mechanism, similar to the transposition of conjugative transposons in Gram-positive bacteria. Class II insertions were characterized by...

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