Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The clinicopathological features and possible physiological mechanisms of only the EGFR-T790M primary mutation in patients with lung adenocarcinoma.

Pathology, research and practice - 1 Jul 2024

Zhao Ping, Xu Liming, Zhu Huijuan, Ding Wei, Tang Hui

Abstract excerpt

BACKGROUND: The treatment of non-small cell lung cancer (NSCLC) patients can be complicated by the presence of the EGFR-T790M mutation. Although primary or secondary EGFR-T790M mutations have been extensively studied worldwide, there are few reports on the clinicopathological characteristics and physiological mechanisms of lung adenocarcinoma (LUAD) with only the EGFR-T790M primary mutation. METHODS: The clinical...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adenocarcinoma of Lung
Share to Aged

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Biomarkers, Tumor

Share to ErbB Receptors

Share to Lung Neoplasms

Share to Mutation

Source

Identifiers

PMID: 38781763
DOI: 10.1016/j.prp.2024.155352

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The clinicopathological features and possible physiological mechanisms of only the EGFR-T790M primary mutation in patients with lung adenocarcinoma.PMID 38781763Read ↗

The clinicopathological features and possible physiological mechanisms of only EGFR-T790M primary mutation in lung adenocarcinoma patientsSemantic proximity Read ↗

Molecular pathology of non-small cell carcinoma.Semantic proximity Read ↗

Putative mechanisms of primary resistance to EGFR-targeted therapies: A retrospective study.Semantic proximity Read ↗

A case of different gene mutations in primary and metastatic lesions after EGFR-TKI treatment of lung cancer.Semantic proximity Read ↗

Different characteristics and survival in non‐small cell lung cancer patients with primary and acquired EGFR T790M mutationSemantic proximity Read ↗

Acquired Mechanism of Crizotinib Resistance in NSCLC with MET Exon 14 SkippingSemantic proximity Read ↗

Whole-exome sequencing identifies key mutated genes in T790M wildtype/cMET-unamplified lung adenocarcinoma with acquired resistance to first-generation EGFR tyrosine kinase inhibitors.Semantic proximity Read ↗

Carcinoma of unknown primary with a lung cancer profile in mediastinal and/or hilar lymph nodes: EGFR-mutant cases and a literature review of actionable drivers.Semantic proximity Read ↗

Acquired crizotinib-resistant pulmonary adenocarcinoma and subsequent primary gallbladder cancer: A case report.Semantic proximity Read ↗

Small-cell carcinoma in the setting of pulmonary adenocarcinoma: new insights in the era of molecular pathology.Semantic proximity Read ↗

Different EGFR gene mutations in two patients with synchronous multiple lung cancers: A case report.Semantic proximity Read ↗

TTC39C may be a new target for the treatment of lung adenocarcinomaSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.