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Article

Epicardial deletion of Sox9 leads to myxomatous valve degeneration and identifies Cd109 as a novel gene associated with valve development

5 Nov 2023

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 37935281
DOI: 10.1016/j.yjmcc.2023.11.002

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Epicardial deletion of Sox9 leads to myxomatous valve degeneration and identifies Cd109 as a novel gene associated with valve developmentPMID 37935281Read ↗

Understanding the role of SOX9 in acquired diseases: lessons from developmentSemantic proximity Read ↗

Essential role of Sox9 in the pathway that controls formation of cardiac valves and septa.Semantic proximity Read ↗

Sox9 deletion causes severe intervertebral disc degeneration characterized by apoptosis, matrix remodeling, and compartment-specific transcriptomic changesSemantic proximity Read ↗

SOX9 in cartilage development and diseaseSemantic proximity Read ↗

Cardiac outflow tract development relies on the complex function of Sox4 and Sox11 in multiple cell typesSemantic proximity Read ↗

SOX9 modulates the expression of key transcription factors required for heart valve developmentSemantic proximity Read ↗

Reduced sox9 function promotes heart valve calcification phenotypes in vivo.Semantic proximity Read ↗

Endocardial Brg1 Represses ADAMTS1 to Maintain the Microenvironment for Myocardial MorphogenesisSemantic proximity Read ↗

Identification and characterization of a novel Schwann and outflow tract endocardial cushion lineage-restricted periostin enhancerSemantic proximity Read ↗

The postnatal role of Sox9 in cartilage.Semantic proximity Read ↗

The transcription factor Sox7 modulates endocardiac cushion formation contributed to atrioventricular septal defect through Wnt4/Bmp2 signalingSemantic proximity Read ↗

Mmp15 is a direct target of Snai1 during endothelial to mesenchymal transformation and endocardial cushion developmentSemantic proximity Read ↗

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