Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

CaV3.2 (CACNA1H) in Primary Aldosteronism.

Handbook of experimental pharmacology - 1 Jan 2023

Dinh Hoang An, Stölting Gabriel, Scholl Ute I

Abstract excerpt

Aldosterone is a steroid hormone produced in the zona glomerulosa (ZG) of the adrenal cortex. The most prominent function of aldosterone is the control of electrolyte homeostasis and blood pressure via the kidneys. The primary factors regulating aldosterone synthesis are the serum concentrations of angiotensin II and potassium. The T-type voltage-gated calcium channel CaV3.2 (encoded by CACNA1H) is an important...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Aldosterone
Share to Calcium Channels, T-Type
Share to Calcium Signaling

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Hyperaldosteronism

Share to Hypertension

Share to Mutation

Source

Identifiers

PMID: 37311830
DOI: 10.1007/164_2023_660

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

CaV3.2 (CACNA1H) in Primary Aldosteronism.PMID 37311830Read ↗

Pathogenesis and treatment of primary aldosteronism.Semantic proximity Read ↗

Update on Genetics of Primary AldosteronismSemantic proximity Read ↗

Voltage-gated calcium channels in the human adrenal and primary aldosteronism.Semantic proximity Read ↗

Genetics of Primary Aldosteronism.Semantic proximity Read ↗

Update on the Genetics of Primary Aldosteronism and Aldosterone-Producing Adenomas.Semantic proximity Read ↗

Recurrent gain of function mutation in calcium channel CACNA1H causes early-onset hypertension with primary aldosteronism.Semantic proximity Read ↗

Genetic causes of primary aldosteronismSemantic proximity Read ↗

GENETICS IN ENDOCRINOLOGY: The expanding genetic horizon of primary aldosteronism.Semantic proximity Read ↗

An update on novel mechanisms of primary aldosteronismSemantic proximity Read ↗

Cellular Pathophysiology of Mutant Voltage-Dependent Ca2+ Channel CACNA1H in Primary Aldosteronism.Semantic proximity Read ↗

Enhanced Ca2+ signaling, mild primary aldosteronism, and hypertension in a familial hyperaldosteronism mouse model (Cacna1hM1560V/+ ).Semantic proximity Read ↗

Novel genes in primary aldosteronism.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.