Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Association of Genetic Variants of HLA-DQA1 with Bullous Pemphigoid Induced by Dipeptidyl Peptidase-4 Inhibitors.

The Journal of investigative dermatology - 1 Nov 2023

Ozeki Takeshi, Muramatsu Ken, Yoshimoto Norihiro, Ujiie Inkin, Izumi Kentaro, Iwata Hiroaki, Mushiroda Taisei, Ujiie Hideyuki

Abstract excerpt

Bullous pemphigoid (BP) is the most common autoimmune blistering disorder. Several factors, including an antidiabetic (dipeptidyl peptidase-4 inhibitor [DPP-4i]), have been reported to trigger BP. To identify the genetic variants associated with BP, GWAS and HLA fine-mapping analyses were conducted. The 21 cases of noninflammatory BP induced by DPP-4i (i.e., DPP-4i-induced noninflammatory BP) and 737 controls...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Aged
Share to Case-Control Studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genetic Predisposition to Disease

Share to Genome-Wide Association Study

Share to HLA-DQ alpha-Chains

Share to Pemphigoid, Bullous

Share to Polymorphism, Single Nucleotide

Source

Identifiers

PMID: 37156394
DOI: 10.1016/j.jid.2023.04.017

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Association of Genetic Variants of HLA-DQA1 with Bullous Pemphigoid Induced by Dipeptidyl Peptidase-4 Inhibitors.PMID 37156394Read ↗

Genetic predisposition to bullous pemphigoid.Semantic proximity Read ↗

Identification of two novel bullous pemphigoid- associated alleles, HLA-DQA1*05:05 and -DRB1*07:01, in GermansSemantic proximity Read ↗

HLA-DQB1*03:01 as a Biomarker for Genetic Susceptibility to Bullous Pemphigoid Induced by DPP-4 Inhibitors.Semantic proximity Read ↗

Recent advances in the genetics and innate immune cells of bullous pemphigoidSemantic proximity Read ↗

Genetics and Omics Analysis of Autoimmune Skin Blistering Diseases.Semantic proximity Read ↗

Association of HLA class II alleles and haplotypes with bullous and mucus membrane pemphigoid risk: A systematic review, a meta-analysis and a meta-regression.Semantic proximity Read ↗

Imputation of the major histocompatibility complex region identifies major independent variants associated with bullous pemphigoid and dermatomyositis in Han Chinese.Semantic proximity Read ↗

Clinical Phenotypes and the Clinical Course of Bullous Pemphigoid Receiving Dipeptidyl Pepitidase-4 Inhibitor Treatment: An Analysis of Cases in a Single Japanese Center.Semantic proximity Read ↗

Molecular Basis for Global Incidence of Pemphigoid Diseases and Differences in Phenotypes.Semantic proximity Read ↗

Dipeptidyl peptidase-4 inhibitor-associated bullous pemphigoid, likely triggered by scabies, in a hemodialysis patient with human leukocyte antigen-DQB1*03:01.Semantic proximity Read ↗

Bullous pemphigoid induced by dipeptidyl peptidase-4 inhibitors. Eight cases with clinical and immunological characterization.Semantic proximity Read ↗

Investigation of the predisposing factor of pemphigus and its clinical subtype through a genome-wide association and next generation sequence analysis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.