Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Early Permissiveness of Central Nervous System Cells to Measles Virus Infection Is Determined by Hyperfusogenicity and Interferon Pressure

13 Jan 2023

Abstract excerpt

The cessation of measles virus (MeV) vaccination in more than 40 countries as a consequence of the COVID-19 pandemic is expected to significantly increase deaths due to measles. MeV can infect the central nervous system (CNS) and lead to lethal encephalitis. Substantial part of virus sequences recovered from patients' brain were mutated in the matrix and/or the fusion protein (F). Mutations of the heptad repeat...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to interferon and immune responses
Share to SARS-CoV-2 and COVID-19 Research
Share to Virology and Viral Diseases

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 36680268
DOI: 10.3390/v15010229

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 24 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Early Permissiveness of Central Nervous System Cells to Measles Virus Infection Is Determined by Hyperfusogenicity and Interferon PressurePMID 36680268Read ↗

Type I Interferon Receptor Signaling Drives Selective Permissiveness of Astrocytes and Microglia to Measles Virus during Brain InfectionSemantic proximity Read ↗

Analysis of a Subacute Sclerosing Panencephalitis Genotype B3 Virus from the 2009-2010 South African Measles Epidemic Shows That Hyperfusogenic F Proteins Contribute to Measles Virus Infection in the BrainSemantic proximity Read ↗

The measles virus matrix F50S mutation from a lethal case of subacute sclerosing panencephalitis promotes receptor-independent neuronal spread.Semantic proximity Read ↗

Molecular Features of the Measles Virus Viral Fusion Complex That Favor Infection and Spread in the BrainSemantic proximity Read ↗

Measles Fusion Machinery Is Dysregulated in Neuropathogenic VariantsSemantic proximity Read ↗

Collective fusion activity determines neurotropism of an en bloc transmitted enveloped virus.Semantic proximity Read ↗

Fitness selection of hyperfusogenic measles virus F proteins associated with neuropathogenic phenotypesSemantic proximity Read ↗

Understanding the Measles Virus at 70 Years: From Cellular Mechanisms to Immunisation, 1954-2024Semantic proximity Read ↗

Mutated Measles Virus Matrix and Fusion Protein Influence Viral Titer In Vitro and Neuro-Invasion in Lewis Rat Brain Slice Cultures.Semantic proximity Read ↗

Brain tropism acquisition: The spatial dynamics and evolution of a measles virus collective infectious unit that drove lethal subacute sclerosing panencephalitisSemantic proximity Read ↗

Fitness selection of hyperfusogenic measles virus F proteins associated with neuropathogenic phenotypes.Semantic proximity Read ↗

Cell-to-Cell Measles Virus Spread between Human Neurons Is Dependent on Hemagglutinin and Hyperfusogenic Fusion ProteinSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.