Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Molecular Features of the Measles Virus Viral Fusion Complex That Favor Infection and Spread in the Brain

11 Jun 2021

Abstract excerpt

Measles virus (MeV) infection can cause serious complications in immunocompromised individuals, including measles inclusion body encephalitis (MIBE). In some cases, MeV persistence and subacute sclerosing panencephalitis (SSPE), another severe central nervous system (CNS) complication, develop even in the face of a systemic immune response. Both MIBE and SSPE are relatively rare but lethal. It is unclear how MeV...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Parvovirus B19 Infection Studies
Share to Viral Infections and Immunology Research
Share to Virology and Viral Diseases

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 34061592
DOI: 10.1128/mbio.00799-21

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 17 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Molecular Features of the Measles Virus Viral Fusion Complex That Favor Infection and Spread in the BrainPMID 34061592Read ↗

Fitness selection of hyperfusogenic measles virus F proteins associated with neuropathogenic phenotypesSemantic proximity Read ↗

Fitness selection of hyperfusogenic measles virus F proteins associated with neuropathogenic phenotypes.Semantic proximity Read ↗

Analysis of a Subacute Sclerosing Panencephalitis Genotype B3 Virus from the 2009-2010 South African Measles Epidemic Shows That Hyperfusogenic F Proteins Contribute to Measles Virus Infection in the BrainSemantic proximity Read ↗

Understanding the Measles Virus at 70 Years: From Cellular Mechanisms to Immunisation, 1954-2024Semantic proximity Read ↗

Measles Fusion Machinery Is Dysregulated in Neuropathogenic VariantsSemantic proximity Read ↗

Brain tropism acquisition: The spatial dynamics and evolution of a measles virus collective infectious unit that drove lethal subacute sclerosing panencephalitisSemantic proximity Read ↗

The measles virus matrix F50S mutation from a lethal case of subacute sclerosing panencephalitis promotes receptor-independent neuronal spread.Semantic proximity Read ↗

Collective fusion activity determines neurotropism of an en bloc transmitted enveloped virus.Semantic proximity Read ↗

Clinico-virological investigation of measles complications in immunocompromised pediatric patients.Semantic proximity Read ↗

Measles Virus Fusion Protein: Structure, Function and InhibitionSemantic proximity Read ↗

Early Permissiveness of Central Nervous System Cells to Measles Virus Infection Is Determined by Hyperfusogenicity and Interferon PressureSemantic proximity Read ↗

Structural and Mechanistic Studies of Measles Virus Illuminate Paramyxovirus EntrySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.