Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Adrenomedullin2 stimulates progression of thyroid cancer in mice and humans under nutrient excess conditions.

The Journal of pathology - 1 Nov 2022

Kim Jung Tae, Lim Mi Ae, Lee Seong Eun, Kim Hyun Jung, Koh Hyun Yong, Lee Jeong Ho, Jun Sang Mi, Kim Jin Man, Kim Kun Ho, Shin Hyo Shik, Cho Sun Wook, Kim Koon Soon, Shong Minho, Koo Bon Seok, Kang Yea Eun

Abstract excerpt

Thyroid cancer is associated with genetic alterations, e.g. BRAFV600E , which may cause carcinomatous changes in hormone-secreting epithelial cells. Epidemiological studies have shown that overnutrition is related to the development and progression of cancer. In this study, we attempted to identify the cell nonautonomous factor responsible for the progression of BRAFV600E thyroid cancer under overnutrition...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cyclic AMP-Dependent Protein Kinases
Share to Extracellular Signal-Regulated MAP Kinases

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Hormones

Share to Nutrients

Share to Palmitic Acid

Share to Peptide Hormones

Share to Proto-Oncogene Proteins B-raf

Share to Thyroid Neoplasms

Source

Open in PubMed Open full text

Identifiers

PMID: 36098211
DOI: 10.1002/path.5997
PMCID: PMC9826144

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Adrenomedullin2 stimulates progression of thyroid cancer in mice and humans under nutrient excess conditions.PMID 36098211Read ↗

Fatty acid oxidation drives Acetyl-CoA-dependent H3K9ac reprogramming to promote adaptive resistance to BRAFV600E inhibition in thyroid cancerSemantic proximity Read ↗

Fine-Tuning Lipid Metabolism by Targeting Mitochondria-Associated Acetyl-CoA-Carboxylase 2 in BRAFV600E Papillary Thyroid Carcinoma.Semantic proximity Read ↗

The global burden and trends of thyroid cancer attributable to high BMI, and the causal impact of obesity: A study integrating GBD 1990-2021 and bidirectional Mendelian randomization.Semantic proximity Read ↗

Obesity and thyroid cancer risk.Semantic proximity Read ↗

Interplay of metabolic dysfunction-associated fatty liver disease and papillary thyroid carcinoma: insights from a Chinese cohort.Semantic proximity Read ↗

Discovery of thyrocyte heterogeneity reveals an essential role of Notch signaling in thyroid function and mammalian homeostasisSemantic proximity Read ↗

Multi-Omics Analysis of Fatty Acid Metabolism in Thyroid CarcinomaSemantic proximity Read ↗

Leptin and insulin synergize with PIK3CA mutation to enhance PD-L1 mediated immunosuppression in thyroid cancer.Semantic proximity Read ↗

Obesity Is Associated with BRAFV600E-Mutated Thyroid Cancer.Semantic proximity Read ↗

Thyroid Nodules and ObesitySemantic proximity Read ↗

The paradoxical lean phenotype of hypothyroid mice is marked by increased adaptive thermogenesis in the skeletal muscle.Semantic proximity Read ↗

Autophagy sustains mitochondrial respiration and determines resistance to BRAF V600E inhibition in thyroid carcinoma cellsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.