Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

SARS-CoV-2 T Cell Responses Elicited by COVID-19 Vaccines or Infection Are Expected to Remain Robust against Omicron.

Viruses - 2 Jan 2022

Ahmed Syed Faraz, Quadeer Ahmed Abdul, McKay Matthew R

Abstract excerpt

Omicron, the most recent SARS-CoV-2 variant of concern (VOC), harbours multiple mutations in the spike protein that were not observed in previous VOCs. Initial studies suggest Omicron to substantially reduce the neutralizing capability of antibodies induced from vaccines and previous infection. However, its effect on T cell responses remains to be determined. Here, we assess the effect of Omicron mutations on...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to COVID-19
Share to COVID-19 Vaccines
Share to Epitopes, T-Lymphocyte

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Mutation

Share to SARS-CoV-2

Share to Spike Glycoprotein, Coronavirus

Share to T-Lymphocytes

Share to Viral Proteins

Source

Open in PubMed Open full text

Identifiers

PMID: 35062283
DOI: 10.3390/v14010079
PMCID: PMC8781795

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

SARS-CoV-2 T Cell Responses Elicited by COVID-19 Vaccines or Infection Are Expected to Remain Robust against Omicron.PMID 35062283Read ↗

The hyper-transmissible SARS-CoV-2 Omicron variant exhibits significant antigenic change, vaccine escape and a switch in cell entry mechanismSemantic proximity Read ↗

SARS-CoV-2 spike T cell responses induced upon vaccination or infection remain robust against OmicronSemantic proximity Read ↗

SARS-CoV-2 Omicron is an immune escape variant with an altered cell entry pathwaySemantic proximity Read ↗

T cell responses to SARS-CoV-2 spike cross-recognize OmicronSemantic proximity Read ↗

Extensive cross-reactive T cell epitopes across SARS-CoV-2 Omicron variant spikes with finite immune evasion mutations.Semantic proximity Read ↗

T cell reactivity to the SARS-CoV-2 Omicron variant is preserved in most but not all individualsSemantic proximity Read ↗

T-cell responses to SARS-CoV-2 Omicron spike epitopes with mutations after the third booster dose of an inactivated vaccine.Semantic proximity Read ↗

SARS-CoV-2 Omicron (BA.1 and BA.2) specific novel CD8+ and CD4+ T cell epitopes targeting spike proteinSemantic proximity Read ↗

COVID‐19: Omicron – the latest, the least virulent, but probably not the last variant of concern of SARS‐CoV‐2Semantic proximity Read ↗

"Is Omicron mild"? Testing this narrative with the mutational landscape of its three lineages and response to existing vaccines and therapeutic antibodies.Semantic proximity Read ↗

Spike protein of SARS‐CoV‐2 Omicron (B.1.1.529) variant has a reduced ability to induce the immune responseSemantic proximity Read ↗

Omicron Reactive Multi Protein Specific CD4 T Cells Defines Cellular Immune Response Induced by Inactivated Virus VaccinesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.