Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

An assessment of executive function in two different rat models of attention‐deficit hyperactivity disorder: Spontaneously hypertensive versus Lphn3 knockout rats

24 Aug 2021

Abstract excerpt

Abstract Attention‐deficit/hyperactivity disorder (ADHD) a common neurodevelopmental disorder of childhood and often comorbid with other externalizing disorders (EDs). There is evidence that externalizing behaviors share a common genetic etiology. Recently, a genome‐wide, multigenerational sample linked variants in the Lphn3 gene to ADHD and other externalizing behaviors. Likewise, limited research in animal...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Attention Deficit Hyperactivity Disorder
Share to Functional Brain Connectivity Studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 34427038
DOI: 10.1111/gbb.12767

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

An assessment of executive function in two different rat models of attention‐deficit hyperactivity disorder: Spontaneously hypertensive versus Lphn3 knockout ratsPMID 34427038Read ↗

Auditory Event-Related Potentials in Two Rat Models of Attention-Deficit Hyperactivity Disorder: Evidence of Automatic Attention Deficits in the Spontaneously Hypertensive but Not LPHN3 Knockout RatsSemantic proximity Read ↗

Behavioral and genetic evidence for a novel animal model of Attention-Deficit/Hyperactivity Disorder Predominantly Inattentive SubtypeSemantic proximity Read ↗

Behavioral disinhibition: liability for externalizing spectrum disorders and its genetic and environmental relation to response inhibition across adolescence.Semantic proximity Read ↗

Intra-individual variability in genetic and environmental models of attention-deficit/hyperactivity disorder.Semantic proximity Read ↗

Genome-wide association study of inattention and hyperactivity-impulsivity measured as quantitative traits.Semantic proximity Read ↗

Reversal Learning Phenotypes are Linked with Novel Genetic Loci in Diversity Outbred MiceSemantic proximity Read ↗

Executive Function as an Underlying Mechanism of Alcohol Use, Aggression, and ADHDSemantic proximity Read ↗

The Evolving ADHD Phenotype in the Externalizing Context.Semantic proximity Read ↗

A new mouse model of ADHD for medication development.Semantic proximity Read ↗

The effect of reduced dopamine D4 receptor expression in the 5-choice continuous performance task: Separating response inhibition from premature responding.Semantic proximity Read ↗

A comparison of phenylketonuria with attention deficit hyperactivity disorder: do markedly different aetiologies deliver common phenotypes?Semantic proximity Read ↗

Neuregulin-3 in the mouse medial prefrontal cortex regulates impulsive action.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.