Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Network medicine links SARS-CoV-2/COVID-19 infection to brain microvascular injury and neuroinflammation in dementia-like cognitive impairment

9 Jun 2021

Abstract excerpt

BACKGROUND: Dementia-like cognitive impairment is an increasingly reported complication of SARS-CoV-2 infection. However, the underlying mechanisms responsible for this complication remain unclear. A better understanding of causative processes by which COVID-19 may lead to cognitive impairment is essential for developing preventive and therapeutic interventions. METHODS: In this study, we conducted a...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Dementia and Cognitive Impairment Research
Share to Long-Term Effects of COVID-19
Share to Neuroinflammation and Neurodegeneration Mechanisms

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 34108016
DOI: 10.1186/s13195-021-00850-3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Network medicine links SARS-CoV-2/COVID-19 infection to brain microvascular injury and neuroinflammation in dementia-like cognitive impairmentPMID 34108016Read ↗

Network medicine links SARS-CoV-2/COVID-19 infection to brain microvascular injury and neuroinflammation in dementia-like cognitive impairmentSemantic proximity Read ↗

Single-nucleus RNA sequencing reveals the shared mechanisms inducing cognitive impairment between COVID-19 and Alzheimer’s diseaseSemantic proximity Read ↗

Investigating the Potential Shared Molecular Mechanisms between COVID-19 and Alzheimer’s Disease via Transcriptomic AnalysisSemantic proximity Read ↗

Common molecular signatures between coronavirus infection and Alzheimer’s disease reveal targets for drug developmentSemantic proximity Read ↗

Brain-wide alterations revealed by spatial transcriptomic and proteomic analyses in COVID-19 infectionSemantic proximity Read ↗

Intersection of COVID-19 and Alzheimer's Disease: Genetic Insights and Neuropathological Consequences.Semantic proximity Read ↗

Systemic infections alter cortical transcriptionalsignatures in Alzheimer’s diseaseSemantic proximity Read ↗

Deterioration of neuroimmune homeostasis in Alzheimer’s Disease patients who survive a COVID-19 infectionSemantic proximity Read ↗

Integrated multi-omics analysis reveals common and distinct dysregulated pathways for genetic subtypes of Frontotemporal DementiaSemantic proximity Read ↗

Deciphering The Molecular Pathogenesis Behind Neurological Manifestations of SARS-CoV-2 And Drug Repurposing, A Systems Biology ApproachSemantic proximity Read ↗

FYN, SARS-CoV-2, and IFITM3 in the Neurobiology of Alzheimer’s Disease: A Regulatory Feedback Loop Governing Tau and Aβ PathologySemantic proximity Read ↗

Investigating Molecular Aspects of SARS-CoV-2 Neurological Manifestations, a Systems Biology ApproachSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.