Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Dysfunction of RAB39B‐ Mediated Vesicular Trafficking in Lewy Body Diseases

3 May 2021

Abstract excerpt

Intracellular vesicular trafficking is essential for neuronal development, function, and homeostasis and serves to process, direct, and sort proteins, lipids, and other cargo throughout the cell. This intricate system of membrane trafficking between different compartments is tightly orchestrated by Ras analog in brain (RAB) GTPases and their effectors. Of the 66 members of the RAB family in humans, many have been...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cellular transport and secretion
Share to Neurological disorders and treatments
Share to Parkinson's Disease Mechanisms and Treatments

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 33939203
DOI: 10.1002/mds.28605

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Dysfunction of RAB39B‐ Mediated Vesicular Trafficking in Lewy Body DiseasesPMID 33939203Read ↗

RAB39B's role in membrane traffic, autophagy, and associated neuropathologySemantic proximity Read ↗

The emerging role of Rab GTPases in the pathogenesis of Parkinson's disease.Semantic proximity Read ↗

The Rab3 family proteins in age-related neurodegeneration: unraveling molecular pathways and potential therapeutic targetsSemantic proximity Read ↗

Parkinson's: A Disease of Aberrant Vesicle TraffickingSemantic proximity Read ↗

The RAB39B p.G192R mutation causes X-linked dominant Parkinson's disease.Semantic proximity Read ↗

Rab GTPases, membrane trafficking and diseases.Semantic proximity Read ↗

Rab GTPases: The Key Players in the Molecular Pathway of Parkinson’s DiseaseSemantic proximity Read ↗

Mutations in RAB39B Cause X-Linked Intellectual Disability and Early-Onset Parkinson Disease with α-Synuclein PathologySemantic proximity Read ↗

The LRRK2-RAB axis in regulation of vesicle trafficking and α-synuclein propagationSemantic proximity Read ↗

An Update on the Interplay between LRRK2, Rab GTPases and Parkinson's Disease.Semantic proximity Read ↗

Rab8a dysregulation in Parkinson's disease: A convergence of genetic and molecular pathologies.Semantic proximity Read ↗

The Role of Rab Proteins in Mitophagy: Insights into Neurodegenerative DiseasesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.