Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Rationale for targeting complement in COVID‐19

19 Jun 2020

Abstract excerpt

A novel coronavirus, SARS-CoV-2, has recently emerged in China and spread internationally, posing a health emergency to the global community. COVID-19 caused by SARS-CoV-2 is associated with an acute respiratory illness that varies from mild to the life-threatening acute respiratory distress syndrome (ARDS). The complement system is part of the innate immune arsenal against pathogens, in which many viruses can...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Complement system in diseases
Share to COVID-19 Clinical Research Studies
Share to SARS-CoV-2 and COVID-19 Research

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 32559343
DOI: 10.15252/emmm.202012642

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 24 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Rationale for targeting complement in COVID‐19PMID 32559343Read ↗

Emerging role of complement in COVID-19 and other respiratory virus diseasesSemantic proximity Read ↗

Complement cascade in severe forms of COVID‐19: Recent advances in therapySemantic proximity Read ↗

Complement Inhibition and COVID-19: The Story so FarSemantic proximity Read ↗

Complement control for COVID-19Semantic proximity Read ↗

The complement system: A key player in the host response to infectionsSemantic proximity Read ↗

A complement atlas identifies interleukin-6–dependent alternative pathway dysregulation as a key druggable feature of COVID-19Semantic proximity Read ↗

Complement and coagulation: key triggers of COVID-19–induced multiorgan pathologySemantic proximity Read ↗

Complement dysregulation is a predictive and therapeutically amenable feature of long COVIDSemantic proximity Read ↗

Targeting complement activation in COVID-19Semantic proximity Read ↗

The complement system in COVID-19: friend and foe?Semantic proximity Read ↗

The life-and-death struggle between the complement system and pathogens: Mechanisms of elimination, evasion tactics, and translational potentialSemantic proximity Read ↗

Alternative pathway dysregulation in tissues drives sustained complement activation and predicts outcome across the disease course in COVID‐19Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.