Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

A single-nucleotide polymorphism induced alternative splicing in Tacr3 involves in hypoxic-ischemic brain damage.

Brain research bulletin - 1 Jan 2020

Xue Lu-Lu, Wang Fang, Xiong Liu-Lin, Du Ruo-Lan, Zhou Hao-Li, Zou Yu, Wu Ma-Xiu, Yang Ming-An, Dai Jing, He Man-Xi, Wang Ting-Hua

Abstract excerpt

Single-nucleotide polymorphism (SNP) and Alternative splicing (AS) were found to be implicated in certain diseases, nevertheless, the contributions of mRNA SNPs and AS to pathogenesis in developing rat brains with hypoxic-ischemic encephalopathy (HIE) remained largely vague. Additionally, the disease associated with Tacr3 was normosmic congenital hypogonadotropic hypogonadism, while the relationship between HIE...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alternative Splicing
Share to Animals, Newborn

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Disease Models, Animal

Share to Hypoxia-Ischemia, Brain

Share to Neurons

Share to Rats, Sprague-Dawley

Source

Identifiers

PMID: 31722250
DOI: 10.1016/j.brainresbull.2019.11.001

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

A single-nucleotide polymorphism induced alternative splicing in Tacr3 involves in hypoxic-ischemic brain damage.PMID 31722250Read ↗

Single-nucleotide polymorphism screening and RNA sequencing of key messenger RNAs associated with neonatal hypoxic-ischemia brain damageSemantic proximity Read ↗

Pathogenicity analysis and splicing rescue of a classical splice site variant (c.1343+1G>T) of CNOT1 gene associated with neurodevelopmental disorders.Semantic proximity Read ↗

Genetic and epigenetic contributors and mimickers of phenotypic hypoxic-ischemic encephalopathy (HIE).Semantic proximity Read ↗

Identification of a Novel Splice-Site variant in TACR3 (c.888 + 1G > A) Associated with Asthenozoospermia and Hypogonadotropic Hypogonadism in an Iranian FamilySemantic proximity Read ↗

Association of NOS3 gene variants and clinical contributors of hypoxic-ischemic encephalopathy.Semantic proximity Read ↗

Alternative Splicing and Hypoxia Puzzle in Alzheimer’s and Parkinson’s DiseasesSemantic proximity Read ↗

Exploring the genetic landscape of neurotransmitter alterations in hypoxic-ischemic encephalopathy: A personalized medicine perspectiveSemantic proximity Read ↗

Novel variant in BRAT1 with the lethal neonatal rigidity and multifocal seizure syndrome.Semantic proximity Read ↗

RETRACTED: Exploring causative gene and pathway of sevoflurane-induced anesthetic effects and implication on nervous system diseases based on high-throughput scRNA-seq dataSemantic proximity Read ↗

Identification of Potential Biomarkers and Pathways in Neonatal Hypoxic-Ischemic Brain Injury: Based on Bioinformatics TechnologySemantic proximity Read ↗

Examination of Genetic Variants Revealed from a Rat Model of Brain Ischemia in Patients with Ischemic Stroke: A Pilot Study.Semantic proximity Read ↗

Long non-coding RNA MEG3 alters neuronal apoptosis and is associated with poor prognosis in ischemic stroke: based on basic and prospective studiesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.