Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

LC3-Associated Endocytosis Facilitates β-Amyloid Clearance and Mitigates Neurodegeneration in Murine Alzheimer’s Disease

27 Jun 2019

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alzheimer's disease research and treatments
Share to Neuroinflammation and Neurodegeneration Mechanisms
Share to Tryptophan and brain disorders

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 31257024
DOI: 10.1016/j.cell.2019.05.056

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

LC3-Associated Endocytosis Facilitates β-Amyloid Clearance and Mitigates Neurodegeneration in Murine Alzheimer’s DiseasePMID 31257024Read ↗

LC3-Associated Endocytosis Facilitates β-Amyloid Clearance and Mitigates Neurodegeneration in Murine Alzheimer’s DiseaseSemantic proximity Read ↗

Autophagy Dysfunction in Alzheimer’s Disease: Mechanistic Insights and New Therapeutic OpportunitiesSemantic proximity Read ↗

Dysfunctional autophagy in Alzheimer’s disease: pathogenic roles and therapeutic implicationsSemantic proximity Read ↗

Galectin-3 drives neuroinflammatory amyloid remodeling by stabilizing intermediate Aβ species and altering lysosomal processingSemantic proximity Read ↗

Dying by fire: noncanonical functions of autophagy proteins in neuroinflammation and neurodegenerationSemantic proximity Read ↗

LC3-associated endocytosis and the functions of Rubicon and ATG16L1Semantic proximity Read ↗

Complement 3a induces the synapse loss via C3aR in mitochondria-dependent NLRP3 activating mechanisms during the development and progression of Alzheimer’s diseaseSemantic proximity Read ↗

The dark sides of amyloid in Alzheimer's disease pathogenesisSemantic proximity Read ↗

ESCRT-III Dysfunction Causes Autophagosome Accumulation and NeurodegenerationSemantic proximity Read ↗

Inhibiting the NLRP3 inflammasome with MCC950 promotes non-phlogistic clearance of amyloid-β and cognitive function in APP/PS1 miceSemantic proximity Read ↗

Protective mechanisms against Alzheimer’s Disease in APOE3-Christchurch homozygous astrocytesSemantic proximity Read ↗

Endo-lysosomal pathway and ubiquitin-proteasome system dysfunction in Alzheimer's disease pathogenesisSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.