Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

TRB3 interacts with ERK and JNK and contributes to the proliferation, apoptosis, and migration of lung adenocarcinoma cells

29 Jun 2019

Abstract excerpt

Tribbles homolog 3 (TRB3) has been accounted for regulation of a few cell processes through interaction with other significant proteins. The molecular mechanisms underlying TRB3 in tumorigenesis in lung adenocarcinoma have not been entirely elucidated. The present study is aimed at determining the function and fundamental mechanisms of TRB3 in lung adenocarcinoma progression. TRB3 was highly expressed in A549 and...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autophagy in Disease and Therapy
Share to Endoplasmic Reticulum Stress and Disease
Share to Hepatocellular Carcinoma Treatment and Prognosis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 31256425
DOI: 10.1002/jcp.28993

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

TRB3 interacts with ERK and JNK and contributes to the proliferation, apoptosis, and migration of lung adenocarcinoma cellsPMID 31256425Read ↗

BTN3A2 Expression Is Connected With Favorable Prognosis and High Infiltrating Immune in Lung AdenocarcinomaSemantic proximity Read ↗

BNIP3 in Lung Cancer: To Kill or Rescue?Semantic proximity Read ↗

B7-H3-Induced Signaling in Lung Adenocarcinoma Cell Lines with Divergent Epidermal Growth Factor Receptor Mutation Patterns.Semantic proximity Read ↗

TRIB3-EGFR interaction promotes lung cancer progression and defines a therapeutic target.Semantic proximity Read ↗

FAM83B promotes the invasion of primary lung adenocarcinoma via PI3K/AKT/NF-κB pathwaySemantic proximity Read ↗

Deletion of TRIB3 Disrupts the Tumor Progression Induced by Integrin αvβ3 in Lung CancerSemantic proximity Read ↗

Mechanism of interaction between TM4SF1 and jak2-stat3 signaling pathway in lung cancer cells and expression analysis in non-small cell lung cancerSemantic proximity Read ↗

TFAP2B overexpression contributes to tumor growth and a poor prognosis of human lung adenocarcinoma through modulation of ERK and VEGF/PEDF signalingSemantic proximity Read ↗

miR-19a-3p Facilitates Lung Adenocarcinoma Cell Phenotypes by Inhibiting TEK.Semantic proximity Read ↗

ELK1 Promotes Epithelial‐Mesenchymal Transition and the Progression of Lung Adenocarcinoma by Upregulating B7‐H3Semantic proximity Read ↗

RYBP Inhibits Progression and Metastasis of Lung Cancer by Suppressing EGFR Signaling and Epithelial-Mesenchymal TransitionSemantic proximity Read ↗

Role of STAT3 in lung cancerSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.