Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Assessment of Fatty Liver in Models of Disease Programming.

Methods in molecular biology (Clifton, N.J.) - 1 Jan 2018

Bruce Kimberley D, Jonscher Karen R

Abstract excerpt

Nonalcoholic fatty liver disease (NAFLD) is currently the most common cause of chronic liver disease worldwide and is present in a third of the general population and the majority of individuals with obesity and type 2 diabetes. Importantly, NAFLD can progress to severe nonalcoholic steatohepatitis (NASH), associated with liver failure and hepatocellular carcinoma. Recent research efforts have extensively focused...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Biopsy
Share to Diet, High-Fat
Share to Disease Susceptibility

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Fatty Liver

Share to Immunohistochemistry

Share to Maternal Exposure

Share to Obesity

Share to Prenatal Exposure Delayed Effects

Source

Identifiers

PMID: 29380318
DOI: 10.1007/978-1-4939-7614-0_15

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Assessment of Fatty Liver in Models of Disease Programming.PMID 29380318Read ↗

Developmental origins of nonalcoholic fatty liver disease.Semantic proximity Read ↗

Developmental Programming of NAFLD by Parental ObesitySemantic proximity Read ↗

A systematic analysis of rodent models implicates adipogenesis and innate immunity in pathogenesis of fatty liver diseaseSemantic proximity Read ↗

Longitudinal analysis of a dietary mouse model of non-alcoholic fatty liver disease (NAFLD) and non-alcoholic steatohepatitis (NASH)Semantic proximity Read ↗

Animal models of NASH: getting both pathology and metabolic context right.Semantic proximity Read ↗

Nonalcoholic fatty liver disease: a challenge for pediatricians.Semantic proximity Read ↗

Studying non-alcoholic fatty liver disease with zebrafish: a confluence of optics, genetics, and physiologySemantic proximity Read ↗

Human fatty liver disease: old questions and new insights.Semantic proximity Read ↗

Comparison of the Phenotype and Approach to Pediatric vs Adult Patients With Nonalcoholic Fatty Liver Disease.Semantic proximity Read ↗

Utilization of animal models to investigate nonalcoholic steatohepatitis-associated hepatocellular carcinoma.Semantic proximity Read ↗

Mouse models in non‐alcoholic fatty liver disease and steatohepatitis researchSemantic proximity Read ↗

Metabolic drivers of non-alcoholic fatty liver disease.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.