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Article

Vitamin D treatment during pregnancy prevents autism-related phenotypes in a mouse model of maternal immune activation.

Molecular autism - 1 Jan 2017

Vuillermot Stephanie, Luan Wei, Meyer Urs, Eyles Darryl

Abstract excerpt

BACKGROUND: Prenatal exposure to infection is a recognized environmental risk factor for neuropsychiatric disorders of developmental origins such as autism or schizophrenia. Experimental work in animals indicates that this link is mediated by maternal immune activation (MIA) involving interactions between cytokine-associated inflammatory events, oxidative stress, and other pathophysiological processes such as...

Topics

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Behavior, Animal
Child Development Disorders, Pervasive
Cytokines
Poly I-C
Prenatal Exposure Delayed Effects

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Social Behavior

Stereotyped Behavior

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Open in PubMed Open full text

Identifiers

PMID: 28316773
DOI: 10.1186/s13229-017-0125-0
PMCID: PMC5351212

Genes

MIA
TNF

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026.

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