Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Cellular aspects of gonadal atrophy in Drosophila P-M hybrid dysgenesis.

Developmental biology - 15 Apr 2017

Dorogova Natalia V, Bolobolova Elena Us, Zakharenko Lyudmila P

Abstract excerpt

Gonadal atrophy is the most typical and dramatic manifestation of intraspecific hybrid dysgenesis syndrome leading to sterility in Drosophila melanogaster dysgenic progeny. The P-M system of hybrid dysgenesis is primarily associated with germ cell degeneration during the early stages of Drosophila embryonic development at elevated temperatures. In the present study, we have defined the phase of germ cell death as...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Atrophy
Share to Drosophila melanogaster
Share to Germ Cells

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Gonads

Share to Hybridization, Genetic

Share to Mitochondria

Share to Phenotype

Share to Temperature

Source

Identifiers

PMID: 28283407
DOI: 10.1016/j.ydbio.2017.02.020

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Cellular aspects of gonadal atrophy in Drosophila P-M hybrid dysgenesis.PMID 28283407Read ↗

Myc plays an important role in Drosophila P-M hybrid dysgenesis to eliminate germline cells with genetic damage.Semantic proximity Read ↗

Mitochondrial fission regulates germ cell differentiation by suppressing ROS-mediated activation of Epidermal Growth Factor Signaling in the Drosophila larval testisSemantic proximity Read ↗

Paternal Mitochondrial Destruction after Fertilization Is Mediated by a Common Endocytic and Autophagic Pathway in DrosophilaSemantic proximity Read ↗

Germ cell connectivity enhances cell death in response to DNA damage in the Drosophila testisSemantic proximity Read ↗

Role of mitochondrial remodeling in programmed cell death in Drosophila melanogaster.Semantic proximity Read ↗

The misregulation of mitochondria-associated genes caused by GAGA-factor lack promotes autophagic germ cell death in Drosophila testes.Semantic proximity Read ↗

p53 is required for female germline stem cell maintenance in P-element hybrid dysgenesis.Semantic proximity Read ↗

Drosophila male-sterile mutation emmenthal specifically affects the mitochondrial morphogenesis.Semantic proximity Read ↗

Nuclear export in somatic cyst cells controls cyst cell-germline coordination and germline differentiation in the <i>Drosophila</i> testisSemantic proximity Read ↗

p53 is required for female germline stem cell maintenance in <i>P</i> -element hybrid dysgenesisSemantic proximity Read ↗

Germ cell connectivity enhances cell death in response to DNA damage in <i>Drosophila</i> testisSemantic proximity Read ↗

Blocking cell fusion inhibits age-induced polyploidy and maintains epithelial organization in <i>Drosophila</i>Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.