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Article

Developmental changes in Notch1 and NLE1 expression in a genetic model of absence epilepsy.

Brain structure & function - 1 Aug 2017

Karimzadeh Fariba, Modarres Mousavi Sayed Mostafa, Alipour Fatemeh, Hosseini Ravandi Hassan, Kovac Stjepana, Gorji Ali

Abstract excerpt

Childhood absence epilepsy (CAE) is an epilepsy syndrome with seizures occurring in the early childhood, highlighting that seizures susceptibility in CAE is dependent on brain development. The Notch 1 signalling pathway is important in brain development, yet the role of the Notch1 signalling pathway in CAE remains elusive. We here explored Notch1 and its modulator notchless homologue 1 (NLE1) expression in...

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Share to Antigens, Nuclear
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Glial Fibrillary Acidic Protein

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Source

Identifiers

PMID: 28210849
DOI: 10.1007/s00429-017-1371-9

Genes

NLE1
GFAP

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

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