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Article

Dysfunction of endothelial progenitor cells in hyperlipidemic rats involves the increase of NADPH oxidase derived reactive oxygen species production.

Canadian journal of physiology and pharmacology - 1 May 2017

Li Ting-Bo, Zhang Jie-Jie, Liu Bin, Luo Xiu-Ju, Ma Qi-Lin, Peng Jun

Abstract excerpt

NADPH oxidase (NOX) is a major source of reactive oxygen species (ROS) in the body and it plays a key role in mediation of oxidative injury in the cardiovascular system. The purposes of this study are to evaluate the status of NOX in endothelial progenitor cells (EPCs) of hyperlipidemic rats and to determine whether NOX-derived ROS promotes the dysfunction of EPCs. The rats were fed on a high-fat diet for 8 weeks...

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PMID: 28177697
DOI: 10.1139/cjpp-2016-0142

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