Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Hepatitis C virus depends on E-cadherin as an entry factor and regulates its expression in epithelial-to-mesenchymal transition

13 Jun 2016

Abstract excerpt

Hepatitis C virus (HCV) enters the host cell through interactions with a cascade of cellular factors. Although significant progress has been made in understanding HCV entry, the precise mechanisms by which HCV exploits the receptor complex and host machinery to enter the cell remain unclear. This intricate process of viral entry likely depends on additional yet-to-be-defined cellular molecules. Recently, by...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Hepatitis B Virus Studies
Share to Hepatitis C virus research
Share to Liver Disease Diagnosis and Treatment

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 27298373
DOI: 10.1073/pnas.1602701113

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 9 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Hepatitis C virus depends on E-cadherin as an entry factor and regulates its expression in epithelial-to-mesenchymal transitionPMID 27298373Read ↗

Regulated Entry of Hepatitis C Virus into HepatocytesSemantic proximity Read ↗

Attachment and Postattachment Receptors Important for Hepatitis C Virus Infection and Cell-to-Cell TransmissionSemantic proximity Read ↗

Temporal analysis of hepatitis C virus cell entry with occludin directed blocking antibodies.Semantic proximity Read ↗

CD81 Is a Central Regulator of Cellular Events Required for Hepatitis C Virus Infection of Human HepatocytesSemantic proximity Read ↗

The Tight Junction Proteins Claudin-1, -6, and -9 Are Entry Cofactors for Hepatitis C VirusSemantic proximity Read ↗

Hepatitis C virus entry: beyond receptorsSemantic proximity Read ↗

Infection with hepatitis C virus depends on TACSTD2, a regulator of claudin-1 and occludin highly downregulated in hepatocellular carcinomaSemantic proximity Read ↗

Isolate-dependent use of claudins for cell entry by hepatitis C virusSemantic proximity Read ↗

Selection of a hepatitis C virus with altered entry factor requirements reveals a genetic interaction between the E1 glycoprotein and claudinsSemantic proximity Read ↗

Claudin-6 and Claudin-9 Function as Additional Coreceptors for Hepatitis C VirusSemantic proximity Read ↗

Integrative Functional Genomics of Hepatitis C Virus Infection Identifies Host Dependencies in Complete Viral Replication CycleSemantic proximity Read ↗

Cellular and molecular biology of HCV infection and hepatitisSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.