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Article

Repression of the eIF2α kinase PERK alleviates mGluR-LTD impairments in a mouse model of Alzheimer's disease

14 Feb 2016

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 27103515
DOI: 10.1016/j.neurobiolaging.2016.02.005

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Related research

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Repression of the eIF2α kinase PERK alleviates mGluR-LTD impairments in a mouse model of Alzheimer's diseasePMID 27103515Read ↗

Suppression of the kinase for elongation factor 2 alleviates mGluR-LTD impairments in a mouse model of Alzheimer’s diseaseSemantic proximity Read ↗

Suppression of eIF2α kinases alleviates Alzheimer's disease-related plasticity and memory deficits.Semantic proximity Read ↗

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Fine‐tuning PERK signaling for neuroprotectionSemantic proximity Read ↗

PERK inhibition prevents tau-mediated neurodegeneration in a mouse model of frontotemporal dementia.Semantic proximity Read ↗

Deletion of the eIF2α Kinase GCN2 fails to rescue the memory decline associated with Alzheimer's disease.Semantic proximity Read ↗

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Chronic PERK induction promotes Alzheimer-like neuropathology in Down syndrome: Insights for therapeutic interventionSemantic proximity Read ↗

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