Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The MAOA, COMT, MTHFR and ESR1 gene polymorphisms are associated with the risk of depression in menopausal women.

Maturitas - 1 Feb 2016

Różycka Agata, Słopień Radosław, Słopień Agnieszka, Dorszewska Jolanta, Seremak-Mrozikiewicz Agnieszka, Lianeri Margarita, Maciukiewicz Małgorzata, Warenik-Szymankiewicz Alina, Grzelak Teresa, Kurzawińska Grażyna, Drews Krzysztof, Klejewski Andrzej, Jagodziński Paweł P

Abstract excerpt

OBJECTIVE: The aim of the study was assessment of a possible relationship between the polymorphisms of the candidate genes participating in the etiology of some neurological and psychiatric disorders and the risk of depression in perimenopausal and postmenopausal women. METHODS: A total of 167 (54 perimenopausal and 113 postmenopausal) Caucasian women from western Poland, aged 42-67, were recruited as the patient...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase
Share to Menopause

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Methylenetetrahydrofolate Dehydrogenase (NADP)

Share to Norepinephrine Plasma Membrane Transport Proteins

Share to Receptor, Serotonin, 5-HT1B

Share to Receptor, Serotonin, 5-HT2A

Share to Receptor, Serotonin, 5-HT2C

Share to Tryptophan Hydroxylase

Source

Identifiers

PMID: 26620113
DOI: 10.1016/j.maturitas.2015.10.011

Genes

COMT
ESR1
MAOA
MTHFR
GABRB1
MTHFD1
MTR
TPH1
TPH2

Variants

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The MAOA, COMT, MTHFR and ESR1 gene polymorphisms are associated with the risk of depression in menopausal women.PMID 26620113Read ↗

Polymorphic variants of genes encoding MTHFR, MTR, and MTHFD1 and the risk of depression in postmenopausal women in Poland.Semantic proximity Read ↗

The c.1460C>T polymorphism of MAO-A is associated with the risk of depression in postmenopausal women.Semantic proximity Read ↗

The 5HTT and MAO-A polymorphisms associate with depressive mood and climacteric symptoms in postmenopausal women.Semantic proximity Read ↗

Polymorphisms in CYP17, COMT, and ESR1 genes in women after menopause and association with bone mineral density.Semantic proximity Read ↗

Association of Catechol-O-methyltransferase (COMT Val158Met) with future risk of cardiovascular disease in depressed individuals - a Swedish population-based cohort study.Semantic proximity Read ↗

Genetic variants in the genes of the sex steroid hormone metabolism and depressive symptoms during and after pregnancy.Semantic proximity Read ↗

Association of C677T polymorphism (rs1801133) in MTHFR gene with depression.Semantic proximity Read ↗

The functional Val158Met polymorphism in catechol-O-methyltransferase (COMT) is associated with depression and motivation in men from a Swedish population-based study.Semantic proximity Read ↗

Depression and the role of genes involved in dopamine metabolism and signalling.Semantic proximity Read ↗

Association between the COMT gene and rumination in a Hungarian sample.Semantic proximity Read ↗

Genetic polymorphisms of methylenetetrahydrofolate reductase (MTHFR) gene and susceptibility to depression in Asian population: a systematic meta-analysis.Semantic proximity Read ↗

Potential role of membrane-bound COMT gene polymorphisms in female depression vulnerability.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.