Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

LRP1B, BRD2 and CACNA1D : New Candidate Genes in Fetal Metabolic Programming of Newborns Exposed to Maternal Hyperglycemia

1 Oct 2015

Abstract excerpt

AIM: To assess the associations between gestational diabetes mellitus (GDM) and DNA methylation levels at genes related to energy metabolism. PATIENTS & METHODS: Ten loci were selected from our recent epigenome-wide association study on GDM. DNA methylation levels were quantified by bisulfite pyrosequencing in 80 placenta and cord blood samples (20 exposed to GDM) from an independent birth cohort (Gen3G)....

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Birth, Development, and Health
Share to Gestational Diabetes Research and Management
Share to Pancreatic function and diabetes

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 26586120
DOI: 10.2217/epi.15.72

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

LRP1B, BRD2 and CACNA1D : New Candidate Genes in Fetal Metabolic Programming of Newborns Exposed to Maternal HyperglycemiaPMID 26586120Read ↗

Epigenetic signatures of gestational diabetes mellitus on cord blood methylationSemantic proximity Read ↗

DNA methylation and expression profiles of placenta and umbilical cord blood reveal the characteristics of gestational diabetes mellitus patients and offspringSemantic proximity Read ↗

Gestational diabetes mellitus epigenetically affects genes predominantly involved in metabolic diseasesSemantic proximity Read ↗

Gestational diabetes and maternal obesity are associated with epigenome-wide methylation changes in childrenSemantic proximity Read ↗

Genome-Wide Placental Gene Methylations in Gestational Diabetes Mellitus, Fetal Growth and Metabolic Health Biomarkers in Cord BloodSemantic proximity Read ↗

DNA methylation patterns and predictive models for metabolic disease risk in offspring of gestational diabetes mellitusSemantic proximity Read ↗

Genomics and Epigenomics of Gestational Diabetes Mellitus: Understanding the Molecular Pathways of the Disease PathogenesisSemantic proximity Read ↗

Maternal DNA Methylation Signatures of Gestational Diabetes across all Stages of PregnancySemantic proximity Read ↗

Maternal Gestational Diabetes Mellitus and Newborn DNA Methylation: Findings From the Pregnancy and Childhood Epigenetics ConsortiumSemantic proximity Read ↗

Placental DNA methylation in pregnancies complicated by maternal diabetes and/or obesity: State of the art and research gapsSemantic proximity Read ↗

Maternal genome-wide DNA methylation profiling in gestational diabetes shows distinctive disease-associated changes relative to matched healthy pregnanciesSemantic proximity Read ↗

In-utero exposure to maternal diabetes and DNA methylation alterations in the Next Generation birth cohortSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.