Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

CELF1 is Up-Regulated in Glioma and Promotes Glioma Cell Proliferation by Suppression of CDKN1B

1 Jan 2015

Abstract excerpt

BACKGROUND: As a member of the CELF family, CELF1 (CUG-binding protein 1, CUGBP1) is involved in cardiac and embryonic development, skeletal muscle differentiation and mammary epithelial cell proliferation. CELF1 is also observed in many kinds of cancer and may play a great role in tumorigenesis and deterioration. However, the expression and mechanism of its function in human glioma remain unclear. METHODS: We...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Glioma Diagnosis and Treatment
Share to Microtubule and mitosis dynamics
Share to Neurogenesis and neuroplasticity mechanisms

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 26535026
DOI: 10.7150/ijbs.11344

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

CELF1 is Up-Regulated in Glioma and Promotes Glioma Cell Proliferation by Suppression of CDKN1BPMID 26535026Read ↗

Comprehensive analysis of the relevance of the CELF gene family to prognosis and immune infiltration in brain gliomaSemantic proximity Read ↗

ELF1 upregulates MEIS1 to accelerate proliferation, migration and invasion of glioma cells through the GFI1/FBW7 axisSemantic proximity Read ↗

Identification of the potential oncogenes in glioblastoma based on bioinformatic analysis and elucidation of the underlying mechanismsSemantic proximity Read ↗

Transcription Factor ELF1 Activates MEIS1 Transcription and Then Regulates the GFI1/FBW7 Axis to Promote the Development of GliomaSemantic proximity Read ↗

Expression of far upstream element (FUSE) binding protein 1 in human glioma is correlated with c‐Myc and cell proliferationSemantic proximity Read ↗

Identification key genes influence cell cycle process in glioma by bioinformatics analysisSemantic proximity Read ↗

Association of EFEMP1 gene polymorphisms with the risk of glioma: A hospital-based case-control study in a Chinese Han population.Semantic proximity Read ↗

TGIF1 overexpression promotes glioma progression and worsens patient prognosisSemantic proximity Read ↗

NAP1L1 Interacts with Hepatoma-Derived Growth Factor to Recruit C-Jun Inducing Breast Cancer GrowthSemantic proximity Read ↗

Suppressor of fused (Sufu) represses Gli1 transcription and nuclear accumulation, inhibits glioma cell proliferation, invasion and vasculogenic mimicry, improving glioma chemo-sensitivity and prognosisSemantic proximity Read ↗

Immune responsive gene 1, a novel oncogene, increases the growth and tumorigenicity of gliomaSemantic proximity Read ↗

The COX10-AS1/miR-641/E2F6 feedback loop is involved in the progression of gliomaSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.