Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Expression of far upstream element (FUSE) binding protein 1 in human glioma is correlated with c‐Myc and cell proliferation

17 Dec 2013

Abstract excerpt

Glioma is one of the most common type of primary intracranial tumor. Although great advances have been achieved in treatment of glioma, the underlying molecular mechanisms remain largely unknown. Previous studies demonstrated that FBP1 is a transcriptional regulator of c-Myc and acts as an important prognostic indicator in many cancers. Our study aimed to assess the expression and function of FBP1 in human...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer-related Molecular Pathways
Share to Cellular transport and secretion
Share to Ubiquitin and proteasome pathways

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 24347226
DOI: 10.1002/mc.22114

Provenance

PubMed metadata and Literature Corpus relationships.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Expression of far upstream element (FUSE) binding protein 1 in human glioma is correlated with c‐Myc and cell proliferationPMID 24347226Read ↗

CMIP Promotes Proliferation and Metastasis in Human GliomaSemantic proximity Read ↗

Transcription Factor ELF1 Activates MEIS1 Transcription and Then Regulates the GFI1/FBW7 Axis to Promote the Development of GliomaSemantic proximity Read ↗

CELF1 is Up-Regulated in Glioma and Promotes Glioma Cell Proliferation by Suppression of CDKN1BSemantic proximity Read ↗

ELF1 upregulates MEIS1 to accelerate proliferation, migration and invasion of glioma cells through the GFI1/FBW7 axisSemantic proximity Read ↗

Loss of FUBP1 expression in gliomas predicts FUBP1 mutation and is associated with oligodendroglial differentiation, IDH1 mutation and 1p/19q loss of heterozygositySemantic proximity Read ↗

Forkhead box transcription factors (FOXOs and FOXM1) in glioma: from molecular mechanisms to therapeuticsSemantic proximity Read ↗

The FDX1 methylation regulatory mechanism in the malignant phenotype of glioma.Semantic proximity Read ↗

c-Fos over-expression promotes radioresistance and predicts poor prognosis in malignant gliomaSemantic proximity Read ↗

Suppressor of fused (Sufu) represses Gli1 transcription and nuclear accumulation, inhibits glioma cell proliferation, invasion and vasculogenic mimicry, improving glioma chemo-sensitivity and prognosisSemantic proximity Read ↗

High PGC-1α Expression as a Poor Prognostic Indicator in Intracranial GliomaSemantic proximity Read ↗

Transcriptional factor specificity protein 1 (SP1) promotes the proliferation of glioma cells by up-regulating midkine (MDK)Semantic proximity Read ↗

GPBAR1 Promotes Proliferation and Is Related to Poor Prognosis of High-Grade Glioma via Inducing MAFB ExpressionSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.