Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

[Polarization of neutrophils from patients with asthma, chronic obstructive pulmonary disease and asthma-chronic obstructive pulmonary disease overlap syndrome].

Zhonghua yi xue za zhi - 30 Dec 2014

Deng Fan, Dong Hangming, Zou Mengchen, Zhao Haijin, Cai Chunqing, Cai Shaoxi

Abstract excerpt

OBJECTIVE: To explore the polarization of migration dynamics of neutrophils isolated from patients with asthma, chronic obstructive pulmonary disease (COPD) and asthma-COPD overlap syndrome (ACOS) compared with healthy smoking and non-smoking controls. METHODS: Recruited volunteers were classified as healthy controls, healthy smokers, asthma, COPD and ACOS at Nanfang Hospital from April 2013 to June 2014...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Asthma
Share to STIM1 gene

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Neutrophils

Share to Phenotype

Share to Pulmonary Disease, Chronic Obstructive

Share to Smoking

Source

Identifiers

PMID: 25623308

Genes

STIM1
STIM2

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

[Polarization of neutrophils from patients with asthma, chronic obstructive pulmonary disease and asthma-chronic obstructive pulmonary disease overlap syndrome].PMID 25623308Read ↗

Macrophage Polarization and Functions in Pathogenesis of Chronic Obstructive Pulmonary DiseaseSemantic proximity Read ↗

Crosstalk of Airway Smooth Muscle and Epithelial Cells in Chronic Lung DiseasesSemantic proximity Read ↗

Store-operated Ca2+ entry contributes to the ASM phenotype transition in asthma.Semantic proximity Read ↗

Research progress on the relationship between phenotype and signaling pathways of pulmonary macrophages and asthma.Semantic proximity Read ↗

Smoking-Dependent Reprogramming of Alveolar Macrophage Polarization: Implication for Pathogenesis of Chronic Obstructive Pulmonary DiseaseSemantic proximity Read ↗

Blood Neutrophils In COPD But Not Asthma Exhibit A Primed Phenotype With Downregulated CD62L Expression.Semantic proximity Read ↗

S1P/HDAC1 Signaling Regulates Macrophage Polarization in Patients with Chronic Obstructive Pulmonary DiseaseSemantic proximity Read ↗

IL-6 trans-signalling: howHaemophilussurfs the NET to amplify inflammation in COPDSemantic proximity Read ↗

Study of Blood Eosinophils and Plasma Periostin as Biomarkers for Response of COPD Patients to ICS/LABA TreatmentSemantic proximity Read ↗

Defective formyl peptide receptor 2/3 and annexin A1 expressions associated with M2a polarization of blood immune cells in patients with chronic obstructive pulmonary disease.Semantic proximity Read ↗

Nuclear receptor corepressor 1 deficiency exacerbates asthma by modulating macrophage polarizationSemantic proximity Read ↗

[Macrophage phenotype in induced sputum in asthma subjects].Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.