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Article

Intima-media thickness and endothelial dysfunction in GCK and HNF1A-MODY patients.

European journal of endocrinology - 1 Mar 2015

Szopa Magdalena, Osmenda Grzegorz, Wilk Grzegorz, Matejko Bartłomiej, Skupien Jan, Zapala Barbara, Młynarski Wojciech, Guzik Tomasz, Malecki Maciej T

Abstract excerpt

OBJECTIVE: Mutations in the glucokinase (GCK) gene, along with hepatocyte nuclear factor 1A (HNF1A) gene mutations, are the most frequent cause of maturity-onset diabetes of the young (MODY). GCK-MODY patients are typically characterized by a moderate fasting hyperglycemia; however, little is known about atherosclerosis and intermediate-related phenotypes in these subjects. DESIGN: To examine carotid artery...

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Share to Carotid Intima-Media Thickness
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Source

Identifiers

PMID: 25501962
DOI: 10.1530/EJE-14-0713

Genes

GCK

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

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Intima-media thickness and endothelial dysfunction in GCK and HNF1A-MODY patients.PMID 25501962Read ↗

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