Angiogenically stimulated alternative monocytes maintain their pro-angiogenic and non-inflammatory phenotype in long-term co-cultures with HUVEC.

doi:10.3233/CH-141875

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Back to search

Article

Angiogenically stimulated alternative monocytes maintain their pro-angiogenic and non-inflammatory phenotype in long-term co-cultures with HUVEC.

Clinical hemorheology and microcirculation - 1 Jan 2014

Krüger Anne, Mayer Anke, Roch Toralf, Schulz Christian, Lendlein Andreas, Jung Friedrich

Abstract excerpt

Angiogenically stimulated alternative monocytes (aMO2) could be established as cellular release system accelerating the endothelialization of polymers rendering their surfaces hemocompatibility in a short-term study. However, for their clinical application it is essential that aMO2 do not switch back to the MO1 state sustaining their capability as cellular release system over an extended period of time. We...

Topics

Angiogenesis Inducing Agents
Biocompatible Materials
Cells, Cultured
Coculture Techniques
Cytokines
Epoprostenol
Human Umbilical Vein Endothelial Cells
Humans
Inflammation
Monocytes
Neovascularization, Pathologic
Neovascularization, Physiologic

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

discussions

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Phenotype

Recombinant Proteins

Thromboxane A2

Vascular Endothelial Growth Factor A

Source

Open in PubMed

Identifiers

PMID: 25227195
DOI: 10.3233/CH-141875

Genes

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 31 Aug 2026.