Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Mitochondrial stress extends lifespan in C. elegans through neuronal hormesis.

Experimental gerontology - 1 Aug 2014

Maglioni Silvia, Schiavi Alfonso, Runci Alessandra, Shaik Anjumara, Ventura Natascia

Abstract excerpt

Progressive neuronal deterioration accompanied by sensory functions decline is typically observed during aging. On the other hand, structural or functional alterations of specific sensory neurons extend lifespan in the nematode Caenorhabditis elegans. Hormesis is a phenomenon by which the body benefits from moderate stress of various kinds which at high doses are harmful. Several studies indicate that different...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Caenorhabditis elegans Proteins
Share to Cellular Senescence

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Longevity

Share to Motor Activity

Share to Sensory Receptor Cells

Share to Stress, Physiological

Source

Identifiers

PMID: 24709340
DOI: 10.1016/j.exger.2014.03.026

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Mitochondrial stress extends lifespan in C. elegans through neuronal hormesis.PMID 24709340Read ↗

Surveying Low-Cost Methods to Measure Lifespan and Healthspan in Caenorhabditis elegans.Semantic proximity Read ↗

Neuronal aging: learning from C. elegansSemantic proximity Read ↗

Neuron-type specific aging-rate reveals age decelerating interventions preventing neurodegenerationSemantic proximity Read ↗

Hijacking Cellular Stress Responses to Promote LifespanSemantic proximity Read ↗

Hormesis extends the correlation between stress resistance and life span in long-lived mutants of Caenorhabditis elegans.Semantic proximity Read ↗

Multiple Molecular Pathways to Longevity: Opposing Gene Expression Programs Define Distinct Aging StrategiesSemantic proximity Read ↗

Relationship between increased longevity and stress resistance as assessed through gerontogene mutations in Caenorhabditis elegans.Semantic proximity Read ↗

The paradox of mitochondrial dysfunction and extended longevity.Semantic proximity Read ↗

Using Caenorhabditis elegans as a model for aging and age-related diseases.Semantic proximity Read ↗

Multiple molecular pathways to longevity with opposing gene expression programs defining distinct aging strategies in Caenorhabditis elegans.Semantic proximity Read ↗

Mitochondria hormesis delays aging and associated diseases in Caenorhabditis elegans impacting on key ferroptosis playersSemantic proximity Read ↗

Regulation of lifespan by sensory perception in Caenorhabditis elegans.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.