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Article

CTCF and BORIS in genome regulation and cancer

14 Dec 2013

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 24657531
DOI: 10.1016/j.gde.2013.10.011

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Related research

Semantic proximity does not establish scientific evidence.

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CTCF and BORIS in genome regulation and cancerPMID 24657531Read ↗

The novel BORIS + CTCF gene family is uniquely involved in the epigenetics of normal biology and cancerSemantic proximity Read ↗

BORIS promotes chromatin regulatory interactions in treatment-resistant cancer cellsSemantic proximity Read ↗

The CTCF Paralog BORIS Contributes to the Ovarian Cancer Transcriptional Program by Relaxing CTCF-mediated 3D Genome OrganizationSemantic proximity Read ↗

The epigenetic factor BORIS (CTCFL) controls the androgen receptor regulatory network in ovarian cancerSemantic proximity Read ↗

Genomic Imprinting: CTCF Protects the BoundariesSemantic proximity Read ↗

Incomplete developmental silencing of cancer-testis antigen BORIS in humanized mouse model promotes cancer susceptibilitySemantic proximity Read ↗

The combined action of CTCF and its testis-specific paralog BORIS is essential for spermatogenesisSemantic proximity Read ↗

Hypomethylation of the CTCFL/BORIS promoter and aberrant expression during endometrial cancer progression suggests a role as an Epi-driver gene.Semantic proximity Read ↗

PIN1, the cell cycle and cancerSemantic proximity Read ↗

BORIS/CTCFL epigenetically reprograms clustered CTCF binding sites into alternative transcriptional start sitesSemantic proximity Read ↗

FKBPs in chromatin modification and cancerSemantic proximity Read ↗

Chromosomal instability (CIN) triggers immune evasion and metastatic potential in cancer through rewired STING signallingSemantic proximity Read ↗

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