Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Broad requirement for terminal sialic acid residues and FcγRIIB for the preventive and therapeutic activity of intravenous immunoglobulins in vivo

6 Feb 2014

Abstract excerpt

Intravenous immunoglobulin (IVIg) therapy is widely used to treat a variety of autoimmune diseases including immunothrombocytopenia, chronic inflammatory demyelinating polyneuropathy, and more recently autoimmune skin blistering diseases. Despite this well-documented clinical success, the precise molecular and cellular mechanisms underlying this immunomodulatory activity are discussed controversially. In...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autoimmune Bullous Skin Diseases
Share to Monoclonal and Polyclonal Antibodies Research
Share to Platelet Disorders and Treatments

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 24505033
DOI: 10.1002/eji.201344230

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Broad requirement for terminal sialic acid residues and FcγRIIB for the preventive and therapeutic activity of intravenous immunoglobulins in vivoPMID 24505033Read ↗

B cells and CD 22 are dispensable for the immediate antiinflammatory activity of intravenous immunoglobulins in vivoSemantic proximity Read ↗

Intravenous immunoglobulins--understanding properties and mechanisms.Semantic proximity Read ↗

Mechanisms of immune regulation by IVIGSemantic proximity Read ↗

Immunomodulation by IVIg and the Role of Fc-Gamma Receptors: Classic Mechanisms of Action after all?Semantic proximity Read ↗

Complete FcRn dependence for intravenous Ig therapy in autoimmune skin blistering diseases.Semantic proximity Read ↗

Understanding Fc Receptor Involvement in Inflammatory Diseases: From Mechanisms to New Therapeutic ToolsSemantic proximity Read ↗

Sialylation of anti-histone immunoglobulin G autoantibodies determines their capabilities to participate in the clearance of late apoptotic cellsSemantic proximity Read ↗

Allogeneic B cell immunomodulatory therapy in amyotrophic lateral sclerosisSemantic proximity Read ↗

CD44 Involvement in Autoimmune InflammationsSemantic proximity Read ↗

Colony-Stimulating Factor-1-Dependent Macrophages Are Responsible for IVIG Protection in Antibody-Induced Autoimmune DiseaseSemantic proximity Read ↗

Structural characterization of anti-inflammatory immunoglobulin G Fc proteins.Semantic proximity Read ↗

Glycosylation of immunoglobulin G determines osteoclast differentiation and bone lossSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.