Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The role of EMMPRIN expression in ovarian epithelial carcinomas.

Cell cycle (Georgetown, Tex.) - 1 Sept 2013

Zhao Yang, Chen Shuo, Gou Wen-feng, Niu Zhe-feng, Zhao Shuang, Xiao Li-jun, Takano Yasuo, Zheng Hua-chuan

Abstract excerpt

PURPOSE: Extracellular matrix metalloproteinase inducer (EMMPRIN) was reported to involve in the invasion and metastasis of malignancies by regulating the expression of vascular endothelial growth factor (VEGF) and matrix metalloproteinases (MMPs) in stromal and cancer cells. The study aimed to clarify the role of EMMPRIN expression in tumorigenesis and progression of ovarian epithelial carcinomas. METHODS:...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Carcinogenesis
Share to Carcinoma, Ovarian Epithelial

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Gene Expression Regulation, Neoplastic

Share to Gene Knockdown Techniques

Share to Basigin

Share to Multivariate Analysis

Share to Neoplasms, Glandular and Epithelial

Share to Ovarian Neoplasms

Source

Open in PubMed Open full text

Identifiers

PMID: 23966157
DOI: 10.4161/cc.25950
PMCID: PMC3899202

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The role of EMMPRIN expression in ovarian epithelial carcinomas.PMID 23966157Read ↗

Silencing of Epidermal Growth Factor-like Domain 8 Promotes Proliferation and Cancer Aggressiveness in Human Ovarian Cancer Cells by Activating ERK/MAPK Signaling CascadesSemantic proximity Read ↗

MMPs in ovarian cancer as therapeutic targets.Semantic proximity Read ↗

Role of Nerve Growth Factor (NGF) and miRNAs in Epithelial Ovarian Cancer.Semantic proximity Read ↗

Silencing of MICAL-L2 suppresses malignancy of ovarian cancer by inducing mesenchymal-epithelial transition.Semantic proximity Read ↗

Ovarian epithelial carcinoma tyrosine phosphorylation, cell proliferation, and ezrin translocation are stimulated by interleukin 1alpha and epidermal growth factor.Semantic proximity Read ↗

EFEMP1 promotes ovarian cancer cell growth, invasion and metastasis via activated the AKT pathwaySemantic proximity Read ↗

miR-448 negatively regulates ovarian cancer cell growth and metastasis by targeting CXCL12Semantic proximity Read ↗

Novel oncogenic and chemoresistance-inducing functions of resistin in ovarian cancer cells require miRNAs-mediated induction of epithelial-to-mesenchymal transitionSemantic proximity Read ↗

ROR2 promotes the epithelial-mesenchymal transition by regulating MAPK/p38 signaling pathway in breast cancer.Semantic proximity Read ↗

ERBB2 gene expression silencing involved in ovarian cancer cell migration and invasion through mediating MAPK1/MAPK3 signaling pathway.Semantic proximity Read ↗

Id1 enhances human ovarian cancer endothelial progenitor cell angiogenesis via PI3K/Akt and NF-κB/MMP-2 signaling pathways.Semantic proximity Read ↗

Overexpression of dedicator of cytokinesis I (Dock180) in ovarian cancer correlated with aggressive phenotype and poor patient survival.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.