Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The impact of reactive oxygen species and genetic mitochondrial mutations in Parkinson's disease.

Gene - 10 Dec 2013

Zuo Li, Motherwell Michael S

Abstract excerpt

The exact pathogenesis of Parkinson's disease (PD) is still unknown and proper mechanisms that correspond to the disease remain unidentified. It is understood that PD is age-related; as age increases, the chance of onset responds accordingly. Although there are no current means of curing PD, the understanding of reactive oxygen species (ROS) provides significant insight to possible treatments. Complex I...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Leucine-Rich Repeat Serine-Threonine Protein Kinase-2
Share to Levodopa
Share to Mitochondria

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Parkinson Disease

Share to Protein Kinases

Share to Protein Serine-Threonine Kinases

Share to Reactive Oxygen Species

Share to Ubiquitin-Protein Ligases

Source

Identifiers

PMID: 23954870
DOI: 10.1016/j.gene.2013.07.085

Genes

PINK1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The impact of reactive oxygen species and genetic mitochondrial mutations in Parkinson's disease.PMID 23954870Read ↗

Mitochondrial dysfunction and oxidative stress in Parkinson’s disease: mechanisms, biomarkers, and therapeutic strategiesSemantic proximity Read ↗

Mitochondrial Dysfunction and Oxidative Stress in Parkinson’s DiseaseSemantic proximity Read ↗

Insights on the Correlation between Mitochondrial Dysfunction and the Progression of Parkinson's Disease.Semantic proximity Read ↗

Mitochondrial biology and oxidative stress in Parkinson disease pathogenesisSemantic proximity Read ↗

Significance of Mitochondrial Dysfunction in the Pathogenesis of Parkinson's DiseaseSemantic proximity Read ↗

Oxidative stress and Parkinson’s diseaseSemantic proximity Read ↗

Oxidative modifications, mitochondrial dysfunction, and impaired protein degradation in Parkinson's disease: how neurons are lost in the Bermuda triangleSemantic proximity Read ↗

Mitochondrial dysfunction and oxidative stress in Parkinson's disease and monogenic parkinsonismSemantic proximity Read ↗

Insights into the Pathogenesis of Neurodegenerative Diseases: Focus on Mitochondrial Dysfunction and Oxidative StressSemantic proximity Read ↗

Oxidative Stress in Genetic Mouse Models of Parkinson’s DiseaseSemantic proximity Read ↗

Mitochondrial Contribution to Parkinson's Disease PathogenesisSemantic proximity Read ↗

Mitochondrial dysfunction and oxidative stress in induced pluripotent stem cell models of Parkinson's diseaseSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.