Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The vir locus affects the response of Bordetella pertussis to antibiotics: phenotypic tolerance and control of autolysis.

The Journal of infectious diseases - 1 Aug 1990

Tuomanen E, Schwartz J, Sande S

Abstract excerpt

Eradication of Bordetella pertussis from the respiratory tract occurs slowly even when bactericidal antibiotics are used. The rate of killing of B. pertussis was found to be proportional to growth rate: virulent, slowly growing strains were killed over days, while rapidly growing strains (either avirulent or virulent modulated by growth conditions to avirulent) were killed over hours. Minimum inhibitory...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Anti-Bacterial Agents
Share to Bacterial Proteins
Share to Carrier Proteins

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Cell Wall

Share to Microscopy, Electron

Share to Molecular Weight

Share to Phenotype

Share to Virulence

Source

Identifiers

PMID: 2373881
DOI: 10.1093/infdis/162.2.560

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The vir locus affects the response of Bordetella pertussis to antibiotics: phenotypic tolerance and control of autolysis.PMID 2373881Read ↗

Avirulent phenotype promotes Bordetella pertussis adaptation to the intramacrophage environment.Semantic proximity Read ↗

A new assay for invasion of HeLa 229 cells by Bordetella pertussis: effects of inhibitors, phenotypic modulation, and genetic alterations.Semantic proximity Read ↗

Mechanisms of bordetella pathogenesisSemantic proximity Read ↗

A versatile, non genetically modified organism (GMO)-based strategy for controlling low-producer mutants in Bordetella pertussis cultures using antigenic modulation.Semantic proximity Read ↗

The effect of erythromycin in Macrolide-Resistant Bordetella pertussis: Inhibitory on Growth, Toxin Expression, and VirulenceSemantic proximity Read ↗

A vir-repressed gene of Bordetella pertussis is required for virulence.Semantic proximity Read ↗

Proteolytically Coordinated Activation of Toxin-Antitoxin ModulesSemantic proximity Read ↗

Fundamental differences in physiology of Bordetella pertussis dependent on the two-component system Bvg revealed by gene essentiality studies.Semantic proximity Read ↗

The virulence factors of<i>Bordetella pertussis</i>: a matter of controlSemantic proximity Read ↗

A novel mechanism of programmed cell death in bacteria by toxin-antitoxin systems corrupts peptidoglycan synthesis.Semantic proximity Read ↗

Genetic and immunological studies on polypeptides encoded by the vir locus of Bordetella pertussis.Semantic proximity Read ↗

Structural Determinants for Antitoxin Identity and Insulation of Cross Talk between Homologous Toxin-Antitoxin Systems.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.