Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Association of endothelial nitric oxide synthase gene polymorphisms with classical risk factors in development of premature coronary artery disease.

Molecular biology reports - 1 Apr 2013

Abdel-Aziz T A, Mohamed R H

Abstract excerpt

Genetic polymorphism of the endothelial nitric oxide synthase (eNOS) affects the pathogenesis of atherosclerosis and associated with premature coronary artery disease (PCAD). We aimed to explore the association between Glu298Asp polymorphism of the eNOS gene and premature CAD in Egyptians, and the possible interaction between this polymorphism and other risk factors. The study population consisted of 116 patients...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Atherosclerosis
Share to Cholesterol
Share to Coronary Artery Disease

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Hypertension

Share to Nitric Oxide Synthase Type III

Share to Triglycerides

Share to White People

Source

Identifiers

PMID: 23269619
DOI: 10.1007/s11033-012-2380-7

Genes

CAD

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Association of endothelial nitric oxide synthase gene polymorphisms with classical risk factors in development of premature coronary artery disease.PMID 23269619Read ↗

The G894T polymorphism on endothelial nitric oxide synthase gene is associated with premature coronary artery disease in a Turkish population.Semantic proximity Read ↗

Association between the eNOS (Glu298Asp) and the RAS genes polymorphisms and premature coronary artery disease in a Turkish population.Semantic proximity Read ↗

Glu298Asp variant of the endothelial nitric oxide synthase gene and acute coronary syndrome or premature coronary artery disease: A systematic review and meta-analysis.Semantic proximity Read ↗

Endothelial nitric oxide synthase and angiotensinogen gene polymorphism in coronary artery diseases in Egypt.Semantic proximity Read ↗

Pathogenesis of premature coronary artery disease: Focus on risk factors and genetic variantsSemantic proximity Read ↗

The association between an endothelial nitric oxide synthase gene polymorphism and coronary heart disease in young people and the underlying mechanism.Semantic proximity Read ↗

Lack of association between endothelial nitric oxide synthase gene polymorphisms and risk of premature coronary artery disease in the Greek population.Semantic proximity Read ↗

Association of polymorphisms of renin angiotensin system and endothelial nitric oxide synthase genes with premature cardiovascular disease in an Iranian population.Semantic proximity Read ↗

Association of polymorphisms of renin angiotensin system and endothelial nitric oxide synthase genes with premature cardiovascular disease in an Iranian populationSemantic proximity Read ↗

The Glu298ASP polymorphism of the endothelial nitric oxide synthase gene is associated with premature ST elevation myocardial infarction in Mexican population.Semantic proximity Read ↗

Relationship between common eNOS gene polymorphisms and predisposition to coronary artery disease: Evidence from a meta-analysis of 155 published association studies.Semantic proximity Read ↗

Association of endothelial nitric oxide synthase (eNOS) gene polymorphism (Glu 298 Asp) with coronary artery disease in subjects from Multan, Pakistan.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.