Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Infant satiety depends on transient expression of cholecystokinin-1 receptors on ependymal cells lining the third ventricle in mice.

The Journal of physiology - 1 Mar 2013

Ozaki Tomoya, Mohammad Shahid, Morioka Eri, Takiguchi Soichi, Ikeda Masayuki

Abstract excerpt

Cholecystokinin (CCK) is a hypothetical controller for suckling and infancy body weight, although the underlying mechanisms remain unclear. Therefore, the present study analysed the mechanisms using mice lacking the CCK-1 receptor (CCK1R-/-). Although CCK1R-/- mice displayed normal weights at birth and adulthood, CCK1R-/- pups had enlarged adipocytes and were overweight from the first to second week after birth,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adipocytes
Share to Animal Nutritional Physiological Phenomena

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Animals, Suckling

Share to Chemokines, CC

Share to Injections, Intraventricular

Share to Microinjections

Share to Overweight

Share to Proto-Oncogene Proteins c-fos

Source

Open in PubMed Open full text

Identifiers

PMID: 23266937
DOI: 10.1113/jphysiol.2012.247676
PMCID: PMC3607872

Genes

CCK

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Infant satiety depends on transient expression of cholecystokinin-1 receptors on ependymal cells lining the third ventricle in mice.PMID 23266937Read ↗

Early ingestive experience with a high-fat diet tunes satiation and nutrient-specific appetitive behaviorsSemantic proximity Read ↗

A single postnatal injection of oxytocin rescues the lethal feeding behaviour in mouse newborns deficient for the imprinted Magel2 gene.Semantic proximity Read ↗

Enterostatin deficiency increases serum cholesterol but does not influence growth and food intake in mice.Semantic proximity Read ↗

Loss of enteroendocrine cells in mice alters lipid absorption and glucose homeostasis and impairs postnatal survival.Semantic proximity Read ↗

Prolactin maintains the neonatal phenotype of enterocytes during lactation in newborn mice.Semantic proximity Read ↗

Feeding and stress interact through the serotonin 2C receptor in developing mice.Semantic proximity Read ↗

Postnatal<i>Sim1</i>Deficiency Causes Hyperphagic Obesity and Reduced<i>Mc4r</i>and<i>Oxytocin</i>ExpressionSemantic proximity Read ↗

Functional synergy between cholecystokinin receptors CCKAR and CCKBR in mammalian brain development.Semantic proximity Read ↗

Neuropeptide Y2 Receptor (NPY2R) Expression in Saliva Predicts Feeding Immaturity in the Premature NeonateSemantic proximity Read ↗

Metabolic insights from a GHSR-A203E mutant mouse model.Semantic proximity Read ↗

Development of Leptin‐Sensitive CircuitsSemantic proximity Read ↗

Nutrient-induced changes in the phenotype and function of the enteric nervous system.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.