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Article

Forkhead box protein 3 (FOXP3) hypermethylation is associated with diesel exhaust exposure and risk for childhood asthma

21 Dec 2012

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Share to Asthma and respiratory diseases
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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 23260754
DOI: 10.1016/j.jaci.2012.10.042

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Related research

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Forkhead box protein 3 (FOXP3) hypermethylation is associated with diesel exhaust exposure and risk for childhood asthmaPMID 23260754Read ↗

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Genetic and epigenetic influence on the response to environmental particulate matter.Semantic proximity Read ↗

Inhalation of diesel exhaust and allergen alters human bronchial epithelium DNA methylationSemantic proximity Read ↗

PM2.5 exposure exacerbates allergic rhinitis in mice by increasing DNA methylation in the IFN-γ gene promoter in CD4+T cells via the ERK-DNMT pathwaySemantic proximity Read ↗

Secondhand smoke in combination with ambient air pollution exposure is associated with increasedx CpG methylation and decreased expression of IFN-γ in T effector cells and Foxp3 in T regulatory cells in childrenSemantic proximity Read ↗

Low expression and hypermethylation of FOXP3 in regulatory T cells are associated with asthma in childrenSemantic proximity Read ↗

Association between long-term air pollution exposure and DNA methylation: the REGICOR studySemantic proximity Read ↗

Influence of single gene variants of FOXP3 on allergic asthma predisposition.Semantic proximity Read ↗

Second-hand smoke and NFE2L2 genotype interaction increases paediatric asthma risk and severity.Semantic proximity Read ↗

Genomic analysis suggests higher susceptibility of children to air pollution.Semantic proximity Read ↗

TET1 Protects the Lungs from Diesel Exhaust Particle-Induced Inflammation and Abnormal FunctionSemantic proximity Read ↗

Ambient air pollution impairs regulatory T-cell function in asthmaSemantic proximity Read ↗

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