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Article

RBM25/LUC7L3 Function in Cardiac Sodium Channel Splicing Regulation of Human Heart Failure

31 Aug 2012

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Share to Cardiac electrophysiology and arrhythmias
Share to Cardiomyopathy and Myosin Studies
Share to RNA Research and Splicing

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 22939879
DOI: 10.1016/j.tcm.2012.08.003

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Related research

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Research trail→

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RBM25/LUC7L3 Function in Cardiac Sodium Channel Splicing Regulation of Human Heart FailurePMID 22939879Read ↗

Role of RBM25/LUC7L3 in Abnormal Cardiac Sodium Channel Splicing Regulation in Human Heart FailureSemantic proximity Read ↗

RBM20-Related Cardiomyopathy: Current Understanding and Future OptionsSemantic proximity Read ↗

Splicing factors in the heart: Uncovering shared and unique targetsSemantic proximity Read ↗

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RBM20, a gene for hereditary cardiomyopathy, regulates titin splicing.Semantic proximity Read ↗

RNA Splicing: Regulation and Dysregulation in the Heart.Semantic proximity Read ↗

Cardiac splicing as a diagnostic and therapeutic targetSemantic proximity Read ↗

Therapeutic potential of alternative splicing in cardiovascular diseases.Semantic proximity Read ↗

Long Non-Coding RNAs (lncRNAs) in Heart Failure: A Comprehensive ReviewSemantic proximity Read ↗

A common coding variant in BAG3 protects from heart failure.Semantic proximity Read ↗

The Emerging Role of the RBM20 and PTBP1 Ribonucleoproteins in Heart Development and Cardiovascular Diseases.Semantic proximity Read ↗

Emerging Role for RBM20 and its Splicing Substrates in Cardiac Function and Heart Failure.Semantic proximity Read ↗

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