Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

An environmental analysis of genes associated with schizophrenia: hypoxia and vascular factors as interacting elements in the neurodevelopmental model.

Molecular psychiatry - 1 Dec 2012

Schmidt-Kastner R, van Os J, Esquivel G, Steinbusch H W M, Rutten B P F

Abstract excerpt

Investigating and understanding gene-environment interaction (G × E) in a neurodevelopmentally and biologically plausible manner is a major challenge for schizophrenia research. Hypoxia during neurodevelopment is one of several environmental factors related to the risk of schizophrenia, and links...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Brain
Share to Cardiovascular Physiological Phenomena
Share to Databases, Genetic

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to DNA Copy Number Variations

Share to Gene-Environment Interaction

Share to Hypoxia-Ischemia, Brain

Share to Models, Neurological

Share to Schizophrenia

Source

Identifiers

PMID: 22290124
DOI: 10.1038/mp.2011.183

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-22290124-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

An environmental analysis of genes associated with schizophrenia: hypoxia and vascular factors as interacting elements in the neurodevelopmental model.PMID 22290124Read ↗

Serious obstetric complications interact with hypoxia-regulated/vascular-expression genes to influence schizophrenia risk.Semantic proximity Read ↗

Gene-environment interaction and covariation in schizophrenia: the role of obstetric complications.Semantic proximity Read ↗

Functional genomics indicate that schizophrenia may be an adult vascular-ischemic disorder.Semantic proximity Read ↗

Placental gene expression mediates the interaction between obstetrical history and genetic risk for schizophreniaSemantic proximity Read ↗

A prospective cohort study of genetic and perinatal influences in the etiology of schizophrenia.Semantic proximity Read ↗

An exploratory model for G x E interaction on hippocampal volume in schizophrenia; obstetric complications and hypoxia-related genes.Semantic proximity Read ↗

Convergence of placenta biology and genetic risk for schizophrenia.Semantic proximity Read ↗

On the nature and mechanisms of obstetric influences in schizophrenia: a review and synthesis of epidemiologic studiesSemantic proximity Read ↗

Cerebral ischemia-induced genes are increased in acute schizophrenia: an opportunity for clinical translation of genomic research findingsSemantic proximity Read ↗

Identification of a Hypoxia-Related Signature as Candidate Detector for Schizophrenia Based on Genome-Wide Gene Expression.Semantic proximity Read ↗

Molecular genetic gene-environment studies using candidate genes in schizophrenia: a systematic review.Semantic proximity Read ↗

Schizophrenia: genes and environment.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.