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Article

Age- and gender-dependent obesity in individuals with 16p11.2 deletion.

Journal of genetics and genomics = Yi chuan xue bao - 20 Sept 2011

Yu Yongguo, Zhu Haitao, Miller David T, Gusella James F, Platt Orah S, Wu Bai-Lin, Shen Yiping

Abstract excerpt

Recurrent genomic imbalances at 16p11.2 are genetic risk factors of variable penetrance for developmental delay and autism. Recently, 16p11.2 (chr16:29.5 Mb-30.1 Mb) deletion has also been detected in individuals with early-onset severe obesity. The penetrance of 16p11.2 deletion as a genetic risk factor for obesity is unknown. We evaluated the growth and body mass characteristics of 28 individuals with 16p11.2...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Source

Identifiers

PMID: 21930099
DOI: 10.1016/j.jgg.2011.08.003

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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