Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Activating transcription factor 3 regulates survivability and migration of vascular smooth muscle cells

1 Jan 2011

Abstract excerpt

Activating transcription factor 3 (ATF3) is a member of the ATF/CREB (CAMP responsive element binding protein) family of transcription factors. The expression and the function of ATF3 in vascular smooth muscle cells (VSMCs) remain unknown. The aim of this work is to determine the expression and possible function of ATF3 in VSMCs. We found that VSMCs expressed ATF3, and expression of ATF3 in VSMCs was induced by a...

Topics

Join the communities discussing this publication.

Nuclear Receptors and Signaling
Renin-Angiotensin System Studies
RNA regulation and disease

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 21280179
DOI: 10.1002/iub.416

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026.

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

0

discussions

0

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Activating transcription factor 3 regulates survivability and migration of vascular smooth muscle cellsPMID 21280179Read ↗

Spatiotemporal ATF3 Expression Determines VSMC Fate in Abdominal Aortic AneurysmSemantic proximity Read ↗

Research on the Correlation Between Activating Transcription Factor 3 Expression in the Human Coronary Artery and Atherosclerotic Plaque StabilitySemantic proximity Read ↗

The Dual Roles of Activating Transcription Factor 3 (ATF3) in Inflammation, Apoptosis, Ferroptosis, and Pathogen Infection ResponsesSemantic proximity Read ↗

MIA SH3 Domain ER Export Factor 3 Deficiency Prevents Neointimal Formation by Restoring BAT-Like PVAT and Decreasing VSMC Proliferation and Migration.Semantic proximity Read ↗

GATA3 modulation of mitochondrial oxidative stress inhibits cerebrovascular remodeling-mediated ischemic stroke by suppressing MBVSMC phenotypic transformation.Semantic proximity Read ↗

Activating Transcription Factor 3 Deficiency Promotes Cardiac Hypertrophy, Dysfunction, and Fibrosis Induced by Pressure OverloadSemantic proximity Read ↗

FOXO3a (Forkhead Transcription Factor O Subfamily Member 3a) Links Vascular Smooth Muscle Cell Apoptosis, Matrix Breakdown, Atherosclerosis, and Vascular Remodeling Through a Novel Pathway Involving MMP13 (Matrix Metalloproteinase 13).Semantic proximity Read ↗

Transcriptional regulation of endothelial-to-mesenchymal transition in cardiac fibrosis: role of myocardin-related transcription factor A and activating transcription factor 3.Semantic proximity Read ↗

ATF3 is required for the prevention of cardiomyopathy via the regulation of mitochondrial oxidative stressSemantic proximity Read ↗

FOXO3a in vascular smooth muscle cell apoptosisSemantic proximity Read ↗

BAG3 promotes proliferation and migration of arterial smooth muscle cells by regulating STAT3 phosphorylation in diabetic vascular remodelingSemantic proximity Read ↗

Activating Transcription Factor 3 Confers Protection against Ventilator-induced Lung InjurySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.