Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Regulation of Nm23-H1 and Cell Invasiveness by Kaposi's Sarcoma-Associated Herpesvirus

27 Jan 2011

Abstract excerpt

The Kaposi's sarcoma-associated herpesvirus (KSHV) is the causative agent of Kaposi's sarcoma (KS), and the induction of an invasive cellular phenotype by KSHV following de novo infection is an important pathogenic component mediating tumor progression. The metastasis suppressor gene known as Nm23-H1 regulates tumor cell invasiveness, but whether KSHV itself regulates Nm23-H1 expression or subcellular...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer Mechanisms and Therapy
Share to Chromatin Remodeling and Cancer
Share to Mechanisms of cancer metastasis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 21270158
DOI: 10.1128/jvi.01596-10

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Regulation of Nm23-H1 and Cell Invasiveness by Kaposi's Sarcoma-Associated HerpesvirusPMID 21270158Read ↗

Epigenetic Landscape of Kaposi's Sarcoma-Associated Herpesvirus Genome in Classic Kaposi's Sarcoma TissuesSemantic proximity Read ↗

Molecular biology and pathogenesis of Kaposi sarcoma-associated herpesvirusSemantic proximity Read ↗

Kaposi’s Sarcoma Herpesvirus Genome PersistenceSemantic proximity Read ↗

Insight into the Epigenetics of Kaposi's Sarcoma Associated HerpesvirusSemantic proximity Read ↗

Epigenetic Regulation of Kaposi’s Sarcoma-Associated Herpesvirus LatencySemantic proximity Read ↗

The Latency-Associated Nuclear Antigen, a Multifunctional Protein Central to Kaposi‘s Sarcoma-Associated Herpesvirus LatencySemantic proximity Read ↗

Nucleophosmin Phosphorylation by v-Cyclin-CDK6 Controls KSHV LatencySemantic proximity Read ↗

Kaposi’s Sarcoma-Associated Herpesvirus, the Etiological Agent of All Epidemiological Forms of Kaposi’s SarcomaSemantic proximity Read ↗

Epigenetic Plasticity Enables CNS-Trafficking of EBV-infected B LymphocytesSemantic proximity Read ↗

LINE-1 transposable element is expressed in Kaposi’s sarcoma and regulates gene expression in KSHV-infected cellsSemantic proximity Read ↗

Kaposi’s Sarcoma-Associated Herpesvirus LANA Hitches a Ride on the ChromosomeSemantic proximity Read ↗

H2AX Phosphorylation Is Important for LANA-Mediated Kaposi's Sarcoma-Associated Herpesvirus Episome PersistenceSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.