Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

VGJphi integration and excision mechanisms contribute to the genetic diversity of Vibrio cholerae epidemic strains.

Proceedings of the National Academy of Sciences of the United States of America - 8 Feb 2011

Das Bhabatosh, Bischerour Julien, Barre François-Xavier

Abstract excerpt

Most strains of Vibrio cholerae are not pathogenic or cause only local outbreaks of gastroenteritis. Acquisition of the capacity to produce the cholera toxin results from a lysogenic conversion event due to a filamentous bacteriophage, CTX. Two V. cholerae tyrosine recombinases that normally serve to resolve chromosome dimers, XerC and XerD, promote CTX integration by directly recombining the ssDNA genome of the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bacterial Proteins
Share to Bacteriophages

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Cholera

Share to Chromosomes, Bacterial

Share to Genetic Variation

Share to Genome, Viral

Share to Vibrio cholerae

Share to Virus Integration

Source

Open in PubMed Open full text

Identifiers

PMID: 21262799
DOI: 10.1073/pnas.1017061108
PMCID: PMC3038760

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

VGJphi integration and excision mechanisms contribute to the genetic diversity of Vibrio cholerae epidemic strains.PMID 21262799Read ↗

Filamentous phage integration requires the host recombinases XerC and XerD.Semantic proximity Read ↗

Holliday junction affinity of the base excision repair factor Endo III contributes to cholera toxin phage integration.Semantic proximity Read ↗

Satellite phage TLCφ enables toxigenic conversion by CTX phage through dif site alteration.Semantic proximity Read ↗

Mobile element integration reveals a chromosome dimer resolution system in LegionellalesSemantic proximity Read ↗

The vibrio pathogenicity island of epidemic Vibrio cholerae forms precise extrachromosomal circular excision products.Semantic proximity Read ↗

Anti-phage islands force their target phage to directly mediate island excision and spreadSemantic proximity Read ↗

Pathogenicity Island Cross Talk Mediated by Recombination Directionality Factors Facilitates Excision from the Chromosome.Semantic proximity Read ↗

Molecular insights into the evolutionary pathway of Vibrio cholerae O1 atypical El Tor variants.Semantic proximity Read ↗

Effect of storage and sodium chloride on excision of CTXPhi or pre-CTXPhi and CTXPhi from Vibrio cholerae O139 strains.Semantic proximity Read ↗

FtsK-dependent and -independent pathways of Xer site-specific recombination.Semantic proximity Read ↗

The crystal structure of bacteriophage λ RexA provides novel insights into the DNA binding properties of Rex-like phage exclusion proteins.Semantic proximity Read ↗

Vibrio vulnificus rtxA1 gene recombination generates toxin variants with altered potency during intestinal infection.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.